V. Kuchroo
Immunology · 2002–2021
A researcher in the Science corpus: 40 papers; trusts 24 researchers and is trusted by 29 researchers here, as inferred from the citation graph.
Subfields
- Immunology — 574 citations into it; #115 of 2,160 by trust here, #21 by citations
- Oncology — 208 citations into it; #1,135 of 1,698 by trust here, #258 by citations
- Pathology and Forensic Medicine — 109 citations into it
- Dermatology — 59 citations into it
Whom V. Kuchroo trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts V. Kuchroo overall
Papers from V. Kuchroo's perspective
Ordered by how strongly the researchers V. Kuchroo trusts endorse each paper. Same corpus, a different order for every researcher.
- Interleukin-23 Promotes a Distinct CD4 T Cell Activation State Characterized by the Production of Interleukin-17*
- Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells (Nature)
- IL-23 drives a pathogenic T cell population that induces autoimmune inflammation
- Interleukin 27 negatively regulates the development of interleukin 17–producing T helper cells during chronic inflammation of the central nervous system (Nature Immunology)
- Microbial Lipopeptides Induce the Production of IL-17 in Th Cells1 (Journal of Immunology)
- Interleukin-17 family members and inflammation. (Immunity)
- Selective regulatory function of Socs3 in the formation of IL-17-secreting T cells. (Proceedings of the National Academy of Sciences of the United States of America)
- Regulatory T cell lineage specification by the forkhead transcription factor foxp3. (Immunity)
- Surface phenotype and antigenic specificity of human interleukin 17–producing T helper memory cells (Nature Immunology)
- Toll Pathway-Dependent Blockade of CD4+CD25+ T Cell-Mediated Suppression by Dendritic Cells (Science)
Their papers
- Genetic and Epigenetic Fine-Mapping of Causal Autoimmune Disease Variants (2014) — 1,818 citations
- Deletion of the gene Pip4k2c, a novel phosphatidylinositol kinase, results in hyperactivation of the immune system (2016) — 62 citations
- Spatially organized multicellular immune hubs in human colorectal cancer (2021) — 691 citations
- The Tim-3 ligand galectin-9 negatively regulates T helper type 1 immunity (2005) — 2,029 citations
- Tim-3 inhibits T helper type 1–mediated auto- and alloimmune responses and promotes immunological tolerance (2003) — 741 citations
- Interaction of Tim-3 and Tim-3 ligand regulates T helper type 1 responses and induction of peripheral tolerance (2003) — 677 citations
- TIM3 comes of age as an inhibitory receptor (2019) — 810 citations
- Tim-3 and its role in regulating anti-tumor immunity (2017) — 739 citations
- Lag-3, Tim-3, and TIGIT co-inhibitory receptors with specialized functions in immune regulation (2016) — 1,913 citations
- Single-cell Genomics Unveils Critical Regulators of Th17 cell Pathogenicity (2015) — 552 citations
- Treg cells expressing the co-inhibitory molecule TIGIT selectively inhibit pro-inflammatory Th1 and Th17 cell responses (2014) — 877 citations
- CEACAM1 regulates TIM–3–mediated tolerance and exhaustion (2014) — 650 citations
- Induction of pathogenic Th17 cells by inducible salt sensing kinase SGK1 (2013) — 995 citations
- Regulatory T cells: recommendations to simplify the nomenclature (2013) — 600 citations
- IL-12 Family Cytokines: Immunological Playmakers (2012) — 1,233 citations
- Induction and molecular signature of pathogenic TH17 cells (2012) — 1,152 citations
- Coexpression of Tim-3 and PD-1 identifies a CD8+ T-cell exhaustion phenotype in mice with disseminated acute myelogenous leukemia. (2011) — 659 citations
- Control of TH17 cells occurs in the Small Intestine (2011) — 679 citations
- Upregulation of Tim-3 and PD-1 expression is associated with tumor antigen–specific CD8+ T cell dysfunction in melanoma patients (2010) — 1,304 citations
- Cooperation of Tim-3 and PD-1 in CD8 T-cell exhaustion during chronic viral infection (2010) — 882 citations