Regulation of Fasted Blood Glucose by Resistin

By Ronadip R. Banerjee, S. Rangwala, Jennifer S. Shapiro, A. Rich, Ben Rhoades, Yong Qi, Juan Wang, Michael W. Rajala, A. Pocai, P. Scherer and 5 more. Science, 2004

759 citations recorded by the source corpus.

Subfields: Endocrine and Autonomic Systems, Epidemiology, Physiology

View Paper: https://www.semanticscholar.org/paper/81566d602b75bc1a233e745742982ea5eb9ff9c6

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Endocrine and Autonomic Systems

1. Haiming Cao
2. G. Steinberg
3. J. Oakhill
4. S. Galic
5. P. Scherer
6. Barry J. Goldstein
7. Rosario Scalia
8. G. Hotamışlıgil

In Epidemiology

1. Haiming Cao
2. B. Spiegelman
3. E. Rosen
4. P. Scherer
5. Barry J. Goldstein
6. Rosario Scalia
7. Mitchell A. Lazar
8. M. Trujillo

In Physiology

1. Haiming Cao
2. B. Spiegelman
3. E. Rosen
4. Mitchell A. Lazar
5. P. Scherer
6. G. Steinberg
7. M. Trujillo
8. N. Ouchi

Citations in the corpus

• Metabolic Syndrome and Insulin Resistance: Underlying Causes and Modification by Exercise Training (2013, positive): “In addition to insulin, AS160 is phosphorylated in response to PDGF, activation of nPKCs and adenosine monophosphate kinase (AMPK), and following skeletal muscle contraction (216, 639).”
• Adipokines in inflammation and metabolic disease (2011, background): “, and mice lacking resistin have low blood glucose levels post-fasting owing to low hepatic glucose productio”
• Mechanisms of obesity-associated insulin resistance: many choices on the menu. (2007, background): “Conversely, mice lacking resistin have improved glucose homeostasis (Banerjee et al. 2004).”
• Adipocytokines in Obesity and Metabolic Disease (2014, positive): “Subsequently, it was shown that resistin knockout mice on a high-fat diet have improved glucose metabolism mainly due to reduced glucose production in the liver (Banerjee et al. 2004).”
• Adipocytes as regulators of energy balance and glucose homeostasis (2006, background): “Levels of resistin are elevated in many murine models of obesity, and gain- and loss-of-function studies in rodents support a role for resistin in increasing hepatic glucose outpu”
• AMP-activated protein kinase: ancient energy gauge provides clues to modern understanding of metabolism. (2005, neutral): “Resistin decreases AMPK phosphorylation (Banerjee et al., 2004; Muse et al., 2004) in liver.”
• Adiponectin: A novel adipokine linking adipocytes and vascular function. (2004, positive): “The adipokine resistin, which mediates glycemia in obesity (63), has been shown to promote endothelial cell activation, with increased endothelin-1 transcription and release and increased expression of the adhesion molecule VCAM-1 and the chemotactic protein VCAM-1 (64).”
• Adipokines: the missing link between insulin resistance and obesity. (2008, background): “A recent study observed a decrease in fasting glucose, mproved glucose tolerance and enhanced insulin sensitivity in esistin-gene knockout mice [57].”
• Elevated plasma level of visfatin/pre-B cell colony-enhancing factor in patients with type 2 diabetes mellitus. (2006, background): “Its effects on insulin resistance has been extensively investigated in mice (28, 29), whereas in humans its role in insulin resistance, obesity, and T2DM has been controversial (30– 32).”
• Role of ROS and RNS Sources in Physiological and Pathological Conditions (2016, background): “In this sense, it has been speculated that lipodystrophy induces insulin resistance, mitochondrial dysfunction, and type 2 diabetes [386].”
• Adipose tissue as an endocrine organ. (2010, background): “Conversely, reducing circulating resistin by deleting the resistin gene, infusing resistin antibodies or resistin antisense oligodeoxynucleotides all protect against obesity-induced hyperglycemia primarily by restoring hepatic insulin responsiveness (Steppan et al., 2001; Banerjee et al., 2004; Muse”