Barry J. Goldstein

Epidemiology · 2004

A researcher in the Science corpus: 1 paper; trusts 8 researchers and is trusted by 2 researchers here, as inferred from the citation graph.

Subfields

Whom Barry J. Goldstein trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. David Carling
  2. J. Sowers
  3. N. Ouchi
  4. Toshimasa Yamauchi
  5. S. Kihara
  6. T. Funahashi
  7. I. Shimomura
  8. M. Matsuda
  9. M. Montagnani
  10. Yuji Matsuzawa

Who trusts Barry J. Goldstein overall

  1. H. Fang
  2. R. L. Judd

Papers from Barry J. Goldstein's perspective

Ordered by how strongly the researchers Barry J. Goldstein trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Proteolytic cleavage product of 30-kDa adipocyte complement-related protein increases fatty acid oxidation in muscle and causes weight loss in mice. (Proceedings of the National Academy of Sciences of the United States of America)
  2. Disruption of Adiponectin Causes Insulin Resistance and Neointimal Formation* (Journal of Biological Chemistry)
  3. Adiponectin stimulates glucose utilization and fatty-acid oxidation by activating AMP-activated protein kinase (Nature Medicine)
  4. Regulation of Fasted Blood Glucose by Resistin (Science)
  5. Adipocyte-Derived Plasma Protein Adiponectin Acts as a Platelet-Derived Growth Factor-BB–Binding Protein and Regulates Growth Factor–Induced Common Postreceptor Signal in Vascular Smooth Muscle Cell (Circulation)
  6. Decreased IRS-2 and increased SREBP-1c lead to mixed insulin resistance and sensitivity in livers of lipodystrophic and ob/ob mice. (Molecules and Cells)
  7. Role of Adiponectin in Preventing Vascular Stenosis (Journal of Biological Chemistry)
  8. Globular Adiponectin Protected ob/ob Mice from Diabetes and ApoE-deficient Mice from Atherosclerosis* (Journal of Biological Chemistry)
  9. Adiponectin-induced antiangiogenesis and antitumor activity involve caspase-mediated endothelial cell apoptosis. (Proceedings of the National Academy of Sciences of the United States of America)
  10. Role of AMP-activated protein kinase in mechanism of metformin action. (Journal of Clinical Investigation)

Their papers