Modulating dysfunctional limbic-cortical circuits in depression: towards development of brain-based algorithms for diagnosis and optimised treatment.

By Helen S. Mayberg. British Medical Bulletin, 2003

1,182 citations recorded by the source corpus.

Subfields: Cognitive Neuroscience, Experimental and Cognitive Psychology, Pharmacology

View Paper: https://www.semanticscholar.org/paper/4bbd1c70cc9aaa2d27df90c79c7a98e4ed452a7f

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Citations in the corpus

The developing brain and neural plasticity: Implications for normality, psychopathology, and resilience. (2015, background): “…interactions, psychotherapy, and pharmacotherapy have the capacity to exert a palliative influence on brains that are afflicted with disorders (Black et al., 1998; Cicchetti, 1996; Cicchetti & Posner, 2005; Cicchetti & Sroufe, 2000; Cicchetti & Walker, 2001; Goldapple et al., 2004; Mayberg, 2003).”
Gray matter abnormalities in Major Depressive Disorder: a meta-analysis of voxel based morphometry studies. (2012, background): “These regions are hypoactive in MDD (Mayberg, 2003) and there is also neuropathological and neurochemical evidence showing abnormality in these regions (Hasler et al.”
The link between childhood trauma and depression: insights from HPA axis studies in humans. (2008, positive): “Areas of difference between these groups overlapped with areas identified as risk markers of depression (Mayberg, 2003).”
Whole Brain Resting-State Analysis Reveals Decreased Functional Connectivity in Major Depression (2010, neutral): “Frontiers in Systems Neuroscience www.frontiersin.org September 2010 | Volume 4 | Article 41 | 1 SYSTEMS NEUROSCIENCE Original research article published: 20 September 2010 doi: 10.3389/fnsys.2010.00041 in this disease (Mayberg, 2003; Drevets et al., 2008).”
In search of the emotional self: an fMRI study using positive and negative emotional words. (2003, background): “that the dysfunction of the dorsomedial prefrontal cortex frequently reported in mood disorders (33) may subserve”
Limbic-frontal circuitry in major depression: a path modeling metanalysis (2004, background): “…is considered, at the brain level, the net result of maladaptive functional interactions among a highly integrated network of limbic – cortical regions normally responsible for maintaining homeostatic emotional control in response to cognitive and somatic stress (Mayberg, 2003; McEwen, 2003).”
Disease state prediction from resting state functional connectivity (2009, methodology): “Fifteen in-brain ROIs were selected based on their relevance to MDD (37) (see Table 1).”
The evolution of the cognitive model of depression and its neurobiological correlates. (2008, background): “However, the findings need to be considered within a broader framework, including many brain regions implicated in depression (43, 44).”
Automatic emotion regulation (2005, background): “…of the SCC is necessary for full recovery from unipolar depression (Davidson, 2002; Drevets, 2001; Gotlib et al., 2005; Liotti et al., 2002; Mayberg, 2003; Shin, Kosslyn, McNally, & Alpert, 1997), and fear extinction in healthy participants is associated with progressively greater…”
Frontocingulate Dysfunction in Depression: Towards Biomarkers of Treatment Response (2010, background): “(a) Mayberg et al (1997): Increased pre-treatment rACC metabolism in responders relative to nonresponders (yellow).”
Neurobiological Mechanisms of the Placebo Effect (2005, background): “…amygdala, and insula), and subcortical (caudate/pallidum, thalamus, and brainstem) regions have been described after such diverse treatments as medication, psychotherapy , sleep deprivation, ECT, repetitive transcranial magnetic stimulation ablative surgery, and DBS (for review, see Mayberg, 2003).”
Modulation of cortical-limbic pathways in major depression: treatment-specific effects of cognitive behavior therapy. (2004, background): “These subjective reports may provide important targets for future investigations of the predictive value of patient treatment preferences and their neural correlates.(90)”
Current research trends in early life stress and depression: review of human studies on sensitive periods, gene-environment interactions, and epigenetics. (2012, positive): “generally modified by early life stress and have been implicated in depression risk (Mayberg, 2003) and control of neuroendocrine reactiv-”