The evolution of the cognitive model of depression and its neurobiological correlates.

By A. Beck. American Journal of Psychiatry, 2008

1,511 citations recorded by the source corpus.

Subfields: Behavioral Neuroscience, Clinical Psychology, Experimental and Cognitive Psychology

View Paper: https://www.semanticscholar.org/paper/d3a055c29ee52176b8d79cf22d2298566812715b

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Citations in the corpus

Advances in cognitive theory and therapy: the generic cognitive model. (2014, background): “Given recent research describing the biological correlates of the cognitive model (Beck 2008, 20 Beck · Haigh A nn u.”
An integrative and functional framework for the study of animal emotion and mood (2010, background): “Such processes are implicated in the aetiology of chronic anxiety and depression in humans (Beck 1967, 2008).”
Pattern separation: a common function for new neurons in hippocampus and olfactory bulb (2011, positive): “Finally, perturbed interpretation of ambiguous cueswhencombinedwith a negative response bias may be a predisposing factor in depression (Beck, 2008; Becker andWojtowicz, 2007; Enkel et al., 2010) and overgeneralized autobiographical memory is often associated with depression and PTSD (Sumner et…”
Cognitive therapy vs. medications for depression: Treatment outcomes and neural mechanisms (2008, background): “Consequently, depressed individuals might suffer from decreased control of amygdala activity as a result of deficits in early automatic prefrontal mechanisms and in the higher-level areas that have been implicated in executive control, such as the DLPF”
Self-awareness, self-regulation, and self-transcendence (S-ART): a framework for understanding the neurobiological mechanisms of mindfulness (2012, background): “Exposure, extinction, and reconsolidation are critical mechanisms for reducing habitual anxiety and fear, and in facilitating therapeutic change in past clinical models (Wells and Matthews, 1996; Bishop, 2007; Beck, 2008).”
Negative Interpretation Bias in Individuals with Depressive Symptoms (2012, background): “In summary, although negative interpretation biases have been clinically recorded and accepted as part of depressotypic thinking for decades (Beck 1967, 1976, 1987, 2008; Bower 1981, 1987; Teasdale 1983), online biases have been difficult to assess in interpretation (for reviews see Gotlib and…”
From Stress to Inflammation and Major Depressive Disorder: A Social Signal Transduction Theory of Depression (2014, neutral): “doi:10.1192/bjp.184.6.526 Anisman, H., & Matheson, K. (2005). Stress, depression, and anhedonia: Caveats concerning animal models. Neuroscience and Biobehavioral Reviews, 29, 525–546. doi:10.1016/j.neubiorev.2005.03.007 Anisman, H., & Merali, Z. (2002). Cytokines, stress, and depressive illness. Bra”
A Cognitive Model of Psychological Resilience (2016, background): “Cognitive models of emotional vulnerability propose that maladaptive cognitive biases (which we describe here as biased information-processing) give rise to dysfunctional cognitions which are activated by stressful events (Beck, 1967; 2008; Beck & Clark, 1988).”
Neural mechanisms of the cognitive model of depression (2011, methodology): “In this Review, we synthesize research findings from neuroimaging studies and provide a neurobiological formulation of the cognitive model of depressio”
The influence of negative emotions on pain: Behavioral effects and neural mechanisms (2009, positive): “In clinical depression, for instance, negative mood as the most prominent feature is almost inevitably accompanied by automatic thoughts, cognitive distortions, dysfunctional beliefs, and information processing biases (Beck, 2008).”
Preventing Symptoms of Depression by Teaching Adolescents That People Can Change (2015, positive): “To do this, we build on cognitive theories of depression (Beck, 1976, 2008; Disner, Beevers, Haigh, & Beck, 2011; Gotlib & Joormann, 2010; Lakdawalla, Hankin, & Mermelstein, 2007).”
A Unified Model of Depression (2016, positive): “…mediated by structural and functional alterations in several brain regions/networks, most notably the amygdala (salience network), hippocampus (default-mode network), and prefrontal cortex (executive network) (Beck, 2008; Disner et al., 2011; Drevets, 2000; Hamilton et al., 2013; Mayberg, 1997).”