Modulation of cortical-limbic pathways in major depression: treatment-specific effects of cognitive behavior therapy.
By K. Goldapple, Z. Segal, C. Garson, M. Lau, P. Bieling, Sidney H. Kennedy, Helen S. Mayberg. Archives of General Psychiatry, 2004
1,046 citations recorded by the source corpus.
Subfields: Cellular and Molecular Neuroscience, Cognitive Neuroscience, Pharmacology
View Paper: https://www.semanticscholar.org/paper/e5337ecb0594d1673fac98dceecbde5366b85634
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Cellular and Molecular Neuroscience
- Robin L. Carhart-Harris
- H. Schmidt
- David J. Nutt
- Christine Marcelle Heim
- Helen S. Mayberg
- Anthony Randal McIntosh
- A. Beck
- Dante R. Chialvo
In Cognitive Neuroscience
- Robin L. Carhart-Harris
- Stephan Hamann
- Christine Marcelle Heim
- Helen S. Mayberg
- F. Benedetti
- Katherine Vytal
- Sidney H. Kennedy
- Tor D. Wager
In Pharmacology
- Christine Marcelle Heim
- A. Beck
- Helen S. Mayberg
- C. Nemeroff
- Sidney H. Kennedy
- David A. Seminowicz
- D. Cicchetti
- W. Curtis
Citations in the corpus
- Prefrontal-subcortical pathways mediating successful emotion regulation. (2008, neutral): “Consider that bilateral vlPFC is activated by both positive and negative emotion-induction tasks (often in tandem with subcortical activation; Kober et al., 2008), that it shows heightened activity in major depression (Drevets et al., 1992) that decreases with successful treatment (Brody et al., 199”
- The integrated response of the human cerebro-cerebellar and limbic systems to acupuncture stimulation at ST 36 as evidenced by fMRI (2005, methodology): “…as measured by BOLD fMRI, PET and electrophysiological methods (Ferris et al., 2004; Friston et al., 1991; Frith et al., 1991; Ghatan et al., 1998; Goldapple et al., 2004; Hutchinson et al., 1999; Liotti et al., 2000; Lustig et al., 2003; Raichle et al., 2001; Shulman et al., 1997; Simpson et…”
- The developing brain and neural plasticity: Implications for normality, psychopathology, and resilience. (2015, background): “…interactions, psychotherapy, and pharmacotherapy have the capacity to exert a palliative influence on brains that are afflicted with disorders (Black et al., 1998; Cicchetti, 1996; Cicchetti & Posner, 2005; Cicchetti & Sroufe, 2000; Cicchetti & Walker, 2001; Goldapple et al., 2004; Mayberg, 2003).”
- Posttraumatic stress disorder: a state-of-the-science review. (2006, background): “9—Mayberg et al., 2000a,b; Mayberg et al., 2002; Liotti et al., 2002a,b; Stefurak and Mayberg, 2003; Goldapple et al., 2004a,b).”
- Deep brain stimulation for treatment-resistant depression. (2005, neutral): “On the other hand, the medial frontal (BA10)/ orbital frontal (BA11) decreases and dorsal cingulate (BA24) increases are similar to the change pattern observed with response to cognitive behavioral therapy (Goldapple et al., 2004).”
- Neuroimaging Support for Discrete Neural Correlates of Basic Emotions: A Voxel-based Meta-analysis (2010, methodology): “intervention studies of depression have demonstrated that reduction in depressive symptoms is associated with increased activity in BA 24 (cingulate cortex), when deep brain stimulation or cognitive behavioral therapy is used (Mayberg et al., 2005; Goldapple et al., 2004), and decreased activity in ”
- Limbic-frontal circuitry in major depression: a path modeling metanalysis (2004, background): “Group 2 (Toronto, n = 14; Goldapple et al., 2004) recruited as part of a separate study, received a 15- week outpatient course of cognitive behavioral therapy (CBT).”
- Targeting abnormal neural circuits in mood and anxiety disorders: from the laboratory to the clinic (2007, background): “Based on data that Cg25 is metabolically overactive in treatment-resistant depression and that activity in this area correlates with response to treatmen”
- Neural mechanisms of the cognitive model of depression (2011, background): “As a result, fewer negative stimuli elicit bottom-up reactivity and the burden on cognitive control systems to regulate subcortical regions would also be mitigate”
- Cognitive therapy vs. medications for depression: Treatment outcomes and neural mechanisms (2008, neutral): “have described, using resting-state PET studies, that recovery in CT is associated with decreased, rather than increased, resting DLPFC metabolis”
- The entropic brain: a theory of conscious states informed by neuroimaging research with psychedelic drugs (2014, background): “…psilocybin in brain regions (e.g., the mPFC) and networks (e.g., the DMN) that are over-engaged in depression (Greicius et al., 2007; Berman et al., 2011) but normalized by a range of effective treatments (Goldapple et al., 2004; Mayberg et al., 2005; Kennedy et al., 2007; Deakin et al., 2008).”
- Neurobiological and psychiatric consequences of child abuse and neglect. (2010, background): “These findings can be discussed in the context of the above study byMayberg and coworkers (Goldapple et al., 2004) that suggested that depressed patients who respond to CBT have different functional activation patterns compared to depressed patients who respond to drug therapy.”
- Neurobiological Mechanisms of the Placebo Effect (2005, result): “Contrary to this hypothesis, recent studies of clinical response to either cognitive behavioral therapy (CBT) (Goldapple et al., 2004) or interpersonal psychotherapy (IPT) (Brody et al.”
- The link between childhood trauma and depression: insights from HPA axis studies in humans. (2008, neutral): “Interestingly, there is evidence from PET studies that depressed patients who respond to cognitive behavioral therapy have different brain changes in response to treatment than patients who respond to drug (Goldapple et al., 2004).”