H. McBride

Molecular Biology · 2001–2014

A researcher in the Science corpus: 7 papers; trusts 10 researchers and is trusted by 14 researchers here, as inferred from the citation graph.

Subfields

Whom H. McBride trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. Derek P. Narendra
  2. R. Youle
  3. D. C. Chan

Who trusts H. McBride overall

  1. S. Dikalov
  2. M. Gutierrez
  3. M. Dodson
  4. I. Slaninová
  5. Seon-Yong Jeong
  6. M. Liesa

Papers from H. McBride's perspective

Ordered by how strongly the researchers H. McBride trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Parkin is recruited selectively to impaired mitochondria and promotes their autophagy (Journal of Cell Biology)
  2. Mitochondrial dysfunction in Drosophila PINK1 mutants is complemented by parkin (Nature)
  3. PINK1 Is Selectively Stabilized on Impaired Mitochondria to Activate Parkin (PLoS Biology)
  4. PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1 (Nature Cell Biology)
  5. Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin (Nature)
  6. PINK1-dependent recruitment of Parkin to mitochondria in mitophagy (Proceedings of the National Academy of Sciences of the United States of America)
  7. Drosophila Parkin requires PINK1 for mitochondrial translocation and ubiquitinates Mitofusin (Proceedings of the National Academy of Sciences of the United States of America)
  8. Fission and selective fusion govern mitochondrial segregation and elimination by autophagy (EMBO Journal)
  9. The PINK1/Parkin pathway regulates mitochondrial morphology (Proceedings of the National Academy of Sciences of the United States of America)
  10. PINK1 stabilized by mitochondrial depolarization recruits Parkin to damaged mitochondria and activates latent Parkin for mitophagy (Journal of Cell Biology)

Their papers