H. Ko
Neurology · Neuron · 2009–2019
A researcher in the Science corpus: 7 papers; trusts 13 researchers and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Neurology — 99 citations into it
- Cellular and Molecular Neuroscience — 66 citations into it
- Physiology — 25 citations into it
- Epidemiology — 15 citations into it
Whom H. Ko trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- M. Goedert
- M. Feany
- W. Dauer
- Valina L. Dawson
- T. Dawson
- J. Palop
- A. Cuervo
- B. Finck
- Derek P. Narendra
- M. Dodson
Who trusts H. Ko overall
Papers from H. Ko's perspective
Ordered by how strongly the researchers H. Ko trusts endorse each paper. Same corpus, a different order for every researcher.
- Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin (Nature)
- Parkin is recruited selectively to impaired mitochondria and promotes their autophagy (Journal of Cell Biology)
- Parkin-deficient Mice Exhibit Nigrostriatal Deficits but Not Loss of Dopaminergic Neurons* (Journal of Biological Chemistry)
- Mitochondrial pathology and apoptotic muscle degeneration in Drosophila parkin mutants (Proceedings of the National Academy of Sciences of the United States of America)
- S-Nitrosylation of Parkin Regulates Ubiquitination and Compromises Parkin's Protective Function (Science)
- Familial Parkinson disease gene product, parkin, is a ubiquitin-protein ligase (Nature Genetics)
- A Drosophila model of Parkinson's disease (Nature)
- PINK1-dependent recruitment of Parkin to mitochondria in mitophagy (Proceedings of the National Academy of Sciences of the United States of America)
- PINK1 Is Selectively Stabilized on Impaired Mitochondria to Activate Parkin (PLoS Biology)
- Hereditary Early-Onset Parkinson's Disease Caused by Mutations in PINK1 (Science)
Their papers
- PARIS (ZNF746) Repression of PGC-1α Contributes to Neurodegeneration in Parkinson’s Disease (2011) — 942 citations
- Midbrain-like organoids from human pluripotent stem cells contain functional dopaminergic and neuromelanin producing neurons (2016) — 762 citations
- Transneuronal Propagation of Pathologic α-Synuclein from the Gut to the Brain Models Parkinson's Disease. (2019) — 1,188 citations
- Block of A1 astrocyte conversion by microglia is neuroprotective in models of Parkinson’s disease (2018) — 943 citations
- Pathological α-synuclein transmission initiated by binding lymphocyte-activation gene 3 (2016) — 670 citations
- Genetic Animal Models of Parkinson’s Disease (2010) — 818 citations
- PINK1-dependent recruitment of Parkin to mitochondria in mitophagy (2009) — 1,653 citations