D. Holtzman

Physiology · 2000–2023

A researcher in the Science corpus: 49 papers; trusts 51 researchers and is trusted by 30 researchers here, as inferred from the citation graph.

Subfields

Whom D. Holtzman trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. G. Bu
  2. Christian Haass
  3. Jungsu Kim
  4. Anne M. Fagan
  5. Clifford R. Jack Jr.
  6. G. Shankar
  7. J. Palop
  8. D. Selkoe
  9. H. Braak
  10. Lars Bertram

Who trusts D. Holtzman overall

  1. J. Long
  2. D. Selkoe
  3. Berislav Zlokovic
  4. Joseph E. Gaugler
  5. G. Bu
  6. Clifford R. Jack Jr.
  7. E. Karran
  8. Nava Zisapel
  9. Rudolph E. Tanzi
  10. Heung-Il Suk

Papers from D. Holtzman's perspective

Ordered by how strongly the researchers D. Holtzman trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Molecular, Structural, and Functional Characterization of Alzheimer's Disease: Evidence for a Relationship between Default Activity, Amyloid, and Memory (Journal of Neuroscience)
  2. Imaging brain amyloid in Alzheimer's disease with Pittsburgh Compound‐B (Annals of Neurology)
  3. Neuropathological stageing of Alzheimer-related changes
  4. Transport Pathways for Clearance of Human Alzheimer's Amyloid β-Peptide and Apolipoproteins E and J in the Mouse Central Nervous System (Journal of Cerebral Blood Flow and Metabolism)
  5. Cerebrospinal fluid {beta}-amyloid 42 and tau proteins as biomarkers of Alzheimer-type pathologic changes in the brain. (Archives of Neurology)
  6. ApoE promotes the proteolytic degradation of Abeta. (Neuron)
  7. Human amyloid-β synthesis and clearance rates as measured in cerebrospinal fluid in vivo (Nature Medicine)
  8. Alzheimer's disease: genes, proteins, and therapy. (Physiological Reviews)
  9. Tau Suppression in a Neurodegenerative Mouse Model Improves Memory Function (Science)
  10. Aberrant excitatory neuronal activity and compensatory remodeling of inhibitory hippocampal circuits in mouse models of Alzheimer's disease. (Neuron)

Their papers