Jungsu Kim

Physiology · 2004–2015

A researcher in the Science corpus: 14 papers; trusts 1 researcher and is trusted by 5 researchers here, as inferred from the citation graph.

Subfields

Whom Jungsu Kim trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. D. Holtzman
  2. R. Deane
  3. D. Selkoe
  4. R. Bell
  5. A. Sagare
  6. Berislav Zlokovic

Who trusts Jungsu Kim overall

  1. D. Holtzman
  2. Rudolph E. Tanzi
  3. Berislav Zlokovic
  4. J. Herz
  5. Yadong Huang
  6. G. Bu
  7. Zeinab Breijyeh
  8. Leslie A. Crews
  9. Richard O'Brien

Papers from Jungsu Kim's perspective

Ordered by how strongly the researchers Jungsu Kim trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Transport Pathways for Clearance of Human Alzheimer's Amyloid β-Peptide and Apolipoproteins E and J in the Mouse Central Nervous System (Journal of Cerebral Blood Flow and Metabolism)
  2. Human amyloid-β synthesis and clearance rates as measured in cerebrospinal fluid in vivo (Nature Medicine)
  3. apoE isoform-specific disruption of amyloid beta peptide clearance from mouse brain. (Journal of Clinical Investigation)
  4. Cerebrospinal fluid {beta}-amyloid 42 and tau proteins as biomarkers of Alzheimer-type pathologic changes in the brain. (Archives of Neurology)
  5. ApoE promotes the proteolytic degradation of Abeta. (Neuron)
  6. APOE Predicts Aβ but not Tau Alzheimer’s Pathology in Cognitively Normal Aging (Annals of Neurology)
  7. P-glycoprotein deficiency at the blood-brain barrier increases amyloid-beta deposition in an Alzheimer disease mouse model. (Journal of Clinical Investigation)
  8. Inverse relation between in vivo amyloid imaging load and cerebrospinal fluid Aβ42 in humans (Annals of Neurology)
  9. Neurofibrillary tangles, amyotrophy and progressive motor disturbance in mice expressing mutant (P301L) tau protein (Nature Genetics)
  10. Decreased beta-amyloid1-42 and increased tau levels in cerebrospinal fluid of patients with Alzheimer disease. (Journal of the American Medical Association (JAMA))

Their papers