Christian Haass

Neurology · 2000–2022

A researcher in the Science corpus: 21 papers; trusts 10 researchers and is trusted by 6 researchers here, as inferred from the citation graph.

Subfields

• Neurology — 179 citations into it
• Physiology — 155 citations into it; #558 of 2,276 by trust here, #235 by citations
• Molecular Biology — 142 citations into it; #16,955 of 17,773 by trust here, #4,951 by citations
• Cellular and Molecular Neuroscience — 93 citations into it; #1,953 of 2,998 by trust here, #1,099 by citations
• Pharmacology — 32 citations into it
• Computational Theory and Mathematics — 25 citations into it
• Epidemiology — 25 citations into it

Whom Christian Haass trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. D. Selkoe
2. Raphael Kopan
3. Derek P. Narendra
4. John R. Cirrito
5. P. Lansbury
6. S. Jin
7. M. Lazarou
8. Marcus E. Raichle

Who trusts Christian Haass overall

1. D. Holtzman
2. J. Long
3. D. Puzzo
4. F. D. De Felice
5. J. Herz
6. Tuomas P. J. Knowles
7. E. Mandelkow
8. L. Privitera
9. G. Bu
10. L. Ittner

Papers from Christian Haass's perspective

Ordered by how strongly the researchers Christian Haass trusts endorse each paper. Same corpus, a different order for every researcher.

1. PINK1 Is Selectively Stabilized on Impaired Mitochondria to Activate Parkin (PLoS Biology)
2. Parkin is recruited selectively to impaired mitochondria and promotes their autophagy (Journal of Cell Biology)
3. Mitochondrial dysfunction in Drosophila PINK1 mutants is complemented by parkin (Nature)
4. PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1 (Nature Cell Biology)
5. Familial Parkinson disease gene product, parkin, is a ubiquitin-protein ligase (Nature Genetics)
6. Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin (Nature)
7. Acceleration of oligomerization, not fibrillization, is a shared property of both alpha-synuclein mutations linked to early-onset Parkinson's disease: implications for pathogenesis and therapy. (Proceedings of the National Academy of Sciences of the United States of America)
8. Molecular, Structural, and Functional Characterization of Alzheimer's Disease: Evidence for a Relationship between Default Activity, Amyloid, and Memory (Journal of Neuroscience)
9. Loss of PINK1 Function Promotes Mitophagy through Effects on Oxidative Stress and Mitochondrial Fission* (Journal of Biological Chemistry)
10. Ubiquitinated TDP-43 in Frontotemporal Lobar Degeneration and Amyotrophic Lateral Sclerosis (Science)

Their papers

• Soluble protein oligomers in neurodegeneration: lessons from the Alzheimer's amyloid β-peptide (2007) — 4,838 citations
• ALS‐associated fused in sarcoma (FUS) mutations disrupt Transportin‐mediated nuclear import (2010) — 825 citations
• Amyloidogenic processing of the Alzheimer β-amyloid precursor protein depends on lipid rafts (2003) — 1,148 citations
• Aβ42‐driven cerebral amyloidosis in transgenic mice reveals early and robust pathology (2006) — 991 citations
• Reconstitution of γ-secretase activity (2003) — 820 citations
• The Role of Presenilins in γ-Secretase Activity* (2001) — 104 citations
• Microglia states and nomenclature: A field at its crossroads (2022) — 1,518 citations
• The TREM2-APOE pathway drives the transcriptional phenotype of dysfunctional microglia in neurodegenerative diseases (2017) — 2,442 citations
• TREM2 mutations implicated in neurodegeneration impair cell surface transport and phagocytosis (2014) — 750 citations
• The C9orf72 GGGGCC Repeat Is Translated into Aggregating Dipeptide-Repeat Proteins in FTLD/ALS (2013) — 1,262 citations
• Mitochondrial dysfunction in Parkinson's disease: molecular mechanisms and pathophysiological consequences (2012) — 624 citations
• Trafficking and proteolytic processing of APP. (2012) — 1,069 citations
• Clusters of Hyperactive Neurons Near Amyloid Plaques in a Mouse Model of Alzheimer's Disease (2008) — 1,069 citations
• Endoplasmic reticulum retention of the γ‐secretase complex component Pen2 by Rer1 (2007) — 85 citations
• Secretion of the Notch-1 Aβ-like Peptide during Notch Signaling* (2006) — 93 citations
• Control of Peripheral Nerve Myelination by the ß-Secretase BACE1 (2006) — 740 citations
• Dimerization of β-Site β-Amyloid Precursor Protein-cleaving Enzyme* (2004) — 97 citations
• Identification of Distinct γ-Secretase Complexes with Different APH-1 Variants* (2004) — 164 citations
• Presenilins mediate a dual intramembranous γ‐secretase cleavage of Notch‐1 (2002) — 213 citations
• Presenilin‐dependent γ‐secretase processing of β‐amyloid precursor protein at a site corresponding to the S3 cleavage of Notch (2001) — 460 citations