Neuropathological stageing of Alzheimer-related changes

By H. Braak, E. Braak. 2004

14,259 citations recorded by the source corpus.

Subfields: Cognitive Neuroscience, Physiology, Psychiatry and Mental health

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Citations in the corpus

Visual Feature Attribution Using Wasserstein GANs (2017, methodology): “As is known from the literature [8, 11] the method indicates atrophy in the hippocampi, and general brain atrophy around the ventricles.”
Axonal Pathology in Traumatic Brain Injury (2012, neutral): “(Braak and Braak, 1991, Corsellis, et al., 1973, Dale, et al., 1991, Forman, et al., 2004, Geddes, et al., 1999, Roberts, et al., 1990, Selkoe, 2001, Tokuda, et al., 1991).”
Dystrophic (senescent) rather than activated microglial cells are associated with tau pathology and likely precede neurodegeneration in Alzheimer’s disease (2009, methodology): “To investigate the temporal relationship between microglial deterioration and neurofibrillary degeneration, we took advantage of the predictability of the spread of tau pathology inherent to Braak staging, namely, that neurodegeneration proceeds predictably from the allocortex (entorhinal and hippoc”
The amyloid hypothesis for Alzheimer’s disease: a critical reappraisal (2009, background): “…of Ab (Hardy 2005; MeyerLuehmann et al. 2006) and MAPT (Frost et al. 2009) the reason for both the characteristic neuroanatomy of the disease (Braak and Braak 1991) as well as the reason that the disease seems to become self-propagating once it has started (Oddo et al. 2004, Santacruz et al.…”
Meta-analysis of 74,046 individuals identifies 11 new susceptibility loci for Alzheimer’s disease (2013, background): “Cases were intermediate or high likelihood by NIA/Reagan criteria with moderate to frequent amyloid plaques [5] and neurofibrillary tangle (NFT) Braak stage of III-VI [6, 7].”
Two-year follow-up of amyloid deposition in patients with Alzheimer's disease. (2006, background): “The regional evolution of Alzheimer’s disease pathology in terms of amyloid deposition (amyloid plaques) and neurofibrillary tangles at several stages of the disease was described by Braak and Braak (1991), using post-mortem brain tissue.”
Neuroinflammation as a Common Feature of Neurodegenerative Disorders (2019, background): “Before the formation of NFTs, the hyperphosphorylation of tau favors a dynamic and progressive self-assembly of tau in oligomeric forms and insoluble materials such as PHFs throughout the disease with different degrees of neurotoxicity (Braak and Braak, 1991).”
Alzheimer's disease (2018, methodology): “Previous pathological criteria for AD from the National Institute of Aging and the Reagan Institute combined the CERAD neuritic plaque score with the Braak and Braak NFT staging, deriving three diagnostic categories: high, intermediate and low likelihood.”
Hierarchical feature representation and multimodal fusion with deep learning for AD/MCI diagnosis (2014, neutral): “Those were distributed around a medial temporal lobe (that includes amygdala, hippocampal formation, entorhinal cortex) (Braak and Braak, 1991; Visser et al., 2002; Mosconi, 2005; Lee et al., 2006; Devanand et al., 2007; Burton et al., 2009; Desikan et al., 2009; Ewers et al., 2012; Walhovd et al., ”
Traumatic brain injury: a disease process, not an event. (2010, background): “Ab plaques and neurofibrillary tangles comprised of tau protein are pathological characteristics of AD (Braak and Braak, 1991; Forman et al., 2004).”
The anatomy of memory: an interactive overview of the parahippocampal–hippocampal network (2009, background): “Moreover, the PHR and HF are implicated in various disorders, such as Alzheimer's diseas”