S. Waguri
Molecular Biology · 2005–2021
A researcher in the Science corpus: 10 papers; trusts 4 researchers and is trusted by 6 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 125 citations into it; #13,646 of 17,773 by trust here, #2,080 by citations
- Epidemiology — 103 citations into it; #528 of 2,148 by trust here, #46 by citations
- Cell Biology — 44 citations into it; #1,410 of 2,073 by trust here, #80 by citations
Whom S. Waguri trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- M. Komatsu
- N. Mizushima
- Beth Levine
- Y. Ohsumi
- Zbyszek Otwinowski
- A. Vagin
- D. Klionsky
- Y. Kabeya
- A. Goldberg
- D. Rubinsztein
Who trusts S. Waguri overall
- N. Mizushima
- M. Komatsu
- E. White
- Alec C. Kimmelman
- D. Rubinsztein
- I. Slaninová
- Robin Mathew
- D. Klionsky
- Beth Levine
- L. Galluzzi
Papers from S. Waguri's perspective
Ordered by how strongly the researchers S. Waguri trusts endorse each paper. Same corpus, a different order for every researcher.
- A role for ubiquitin in selective autophagy. (Molecules and Cells)
- The selective autophagy substrate p62 activates the stress responsive transcription factor Nrf2 through inactivation of Keap1 (Nature Cell Biology)
- PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1 (Nature Cell Biology)
- Autophagy fights disease through cellular self-digestion (Nature)
- LC3, GABARAP and GATE16 localize to autophagosomal membrane depending on form-II formation (Journal of Cell Science)
- LC3, a mammalian homologue of yeast Apg8p, is localized in autophagosome membranes after processing (EMBO Journal)
- A ubiquitin-like system mediates protein lipidation (Nature)
- p62/SQSTM1 Is a Target Gene for Transcription Factor NRF2 and Creates a Positive Feedback Loop by Inducing Antioxidant Response Element-driven Gene Transcription* (Journal of Biological Chemistry)
- The mitochondrial permeability transition initiates autophagy in rat hepatocytes (The FASEB Journal)
- Development by self-digestion: molecular mechanisms and biological functions of autophagy. (Developmental Cell)
Their papers
- Homeostatic levels of p62 control cytoplasmic inclusion body formation in autophagy-deficient mice. (2007) — 2,205 citations
- The selective autophagy substrate p62 activates the stress responsive transcription factor Nrf2 through inactivation of Keap1 (2010) — 2,354 citations
- Phosphorylation of p62 activates the Keap1-Nrf2 pathway during selective autophagy. (2013) — 1,109 citations
- Persistent activation of Nrf2 through p62 in hepatocellular carcinoma cells (2011) — 599 citations
- Autophagy-deficient mice develop multiple liver tumors. (2011) — 1,308 citations
- Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1 (2021) — 3,841 citations
- Activation of STING requires palmitoylation at the Golgi (2016) — 635 citations
- Uptake through glycoprotein 2 of FimH+ bacteria by M cells initiates mucosal immune response (2009) — 614 citations
- Loss of autophagy in the central nervous system causes neurodegeneration in mice (2006) — 3,615 citations
- Impairment of starvation-induced and constitutive autophagy in Atg7-deficient mice (2005) — 2,479 citations