T. Senoner
Molecular Biology · 2019
A researcher in the Science corpus: 1 paper; trusts 5 researchers and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 14 citations into it
- Immunology — 12 citations into it
- Cardiology and Cardiovascular Medicine — 9 citations into it
- Nutrition and Dietetics — 9 citations into it
- Biochemistry — 8 citations into it
- Physiology — 8 citations into it
Whom T. Senoner trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- Toren Finkel
- Barry Halliwell
- Rahul Wadke
- S. Liochev
- Rhian M. Touyz
- Michael Ristow
- K. Bedard
- M. Circu
- T. Paravicini
- C. Serhan
Papers from T. Senoner's perspective
Ordered by how strongly the researchers T. Senoner trusts endorse each paper. Same corpus, a different order for every researcher.
- REACTIVE OXYGEN SPECIES, CELLULAR REDOX SYSTEMS AND APOPTOSIS (Free Radical Biology & Medicine)
- The NOX family of ROS-generating NADPH oxidases: physiology and pathophysiology. (Physiological Reviews)
- Free radicals and antioxidants in normal physiological functions and human disease. (International Journal of Biochemistry and Cell Biology)
- Antioxidant and prooxidant properties of flavonoids. (Fitoterapia)
- Adherence to a Mediterranean diet and survival in a Greek population. (New England Journal of Medicine)
- Resolving inflammation: dual anti-inflammatory and pro-resolution lipid mediators (Nature reviews. Immunology)
- Cellular mechanisms and physiological consequences of redox-dependent signalling (Nature reviews. Molecular cell biology)
- Circulating endothelial progenitor cells, vascular function, and cardiovascular risk. (New England Journal of Medicine)
- Macrophage myeloperoxidase regulation by granulocyte macrophage colony-stimulating factor in human atherosclerosis and implications in acute coronary syndromes. (American Journal of Pathology)
- GPR120 is an omega-3 fatty acid receptor mediating potent anti-inflammatory and insulin-sensitizing effects. (Cell)
Their papers
- Oxidative Stress in Cardiovascular Diseases: Still a Therapeutic Target? (2019) — 742 citations