Shengkan Jin

Epidemiology · 2003–2016

A researcher in the Science corpus: 9 papers; trusts 4 researchers and is trusted by 22 researchers here, as inferred from the citation graph.

Subfields

• Epidemiology — 101 citations into it; #88 of 2,148 by trust here, #79 by citations
• Molecular Biology — 88 citations into it; #1,679 of 17,773 by trust here, #1,504 by citations
• Cell Biology — 40 citations into it
• Pharmacology — 13 citations into it

Whom Shengkan Jin trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. N. Mizushima

Who trusts Shengkan Jin overall

1. Beth Levine
2. E. White
3. N. Mizushima
4. Alec C. Kimmelman
5. Guido Kroemer
6. L. Galluzzi
7. Robin Mathew
8. M. Azad
9. D. Klionsky
10. Y. Tsujimoto

Papers from Shengkan Jin's perspective

Ordered by how strongly the researchers Shengkan Jin trusts endorse each paper. Same corpus, a different order for every researcher.

1. Autophagy: from phenomenology to molecular understanding in less than a decade (Nature reviews. Molecular cell biology)
2. Methods for Monitoring Autophagy from Yeast to Human (Autophagy)
3. Autophagy promotes tumor cell survival and restricts necrosis, inflammation, and tumorigenesis. (Cancer Cell)
4. A unified nomenclature for yeast autophagy-related genes. (Developmental Cell)
5. Rheb GTPase is a direct target of TSC2 GAP activity and regulates mTOR signaling. (Genes & Development)
6. Autophagy: process and function. (Genes & Development)
7. Guidelines for the use and interpretation of assays for monitoring autophagy in higher eukaryotes (Autophagy)
8. Regulation of an ATG7-beclin 1 Program of Autophagic Cell Death by Caspase-8 (Science)
9. The role of autophagy during the early neonatal starvation period (Nature)
10. Beclin 1, an autophagy gene essential for early embryonic development, is a haploinsufficient tumor suppressor (Proceedings of the National Academy of Sciences of the United States of America)

Their papers

• Beclin 1, an autophagy gene essential for early embryonic development, is a haploinsufficient tumor suppressor (2003) — 2,229 citations
• The coordinate regulation of the p53 and mTOR pathways in cells. (2005) — 1,244 citations
• Autophagy promotes tumor cell survival and restricts necrosis, inflammation, and tumorigenesis. (2006) — 2,081 citations
• The regulation of AMPK beta1, TSC2, and PTEN expression by p53: stress, cell and tissue specificity, and the role of these gene products in modulating the IGF-1-AKT-mTOR pathways. (2007) — 629 citations
• Autophagy suppresses tumor progression by limiting chromosomal instability. (2007) — 990 citations
• Autophagy mitigates metabolic stress and genome damage in mammary tumorigenesis. (2007) — 882 citations
• Editorial :Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition). DOI: 10.1080/15548627.2015.1100356;WOS:000373595400001; 2-s2.0-85013763791&;PMID: 26799652 (2016) — 1,523 citations
• Adipose-specific deletion of autophagy-related gene 7 (atg7) in mice reveals a role in adipogenesis (2009) — 653 citations
• Guidelines for the use and interpretation of assays for monitoring autophagy in higher eukaryotes (2008) — 2,657 citations