Bryan A. Ballif
Molecular Biology · 2004–2007
A researcher in the Science corpus: 6 papers; trusts 3 researchers and is trusted by 5 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 41 citations into it; #7,258 of 17,773 by trust here, #2,948 by citations
- Physiology — 5 citations into it; #1,034 of 2,276 by trust here, #1,048 by citations
Whom Bryan A. Ballif trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts Bryan A. Ballif overall
- R. Anjum
- David M. Sabatini
- Young Chul Kim
- J. Blenis
- M. Laplante
- David Holcman
- R. Shaw
- Olivier Bénichou
- Chunaram Choudhary
Papers from Bryan A. Ballif's perspective
Ordered by how strongly the researchers Bryan A. Ballif trusts endorse each paper. Same corpus, a different order for every researcher.
- The Mammalian Target of Rapamycin (mTOR) Partner, Raptor, Binds the mTOR Substrates p70 S6 Kinase and 4E-BP1 through Their TOR Signaling (TOS) Motif* (Journal of Biological Chemistry)
- TOR, a central controller of cell growth. (Cell)
- Regulation of elongation factor 2 kinase by p90RSK1 and p70 S6 kinase (EMBO Journal)
- S6K1−/−/S6K2−/− Mice Exhibit Perinatal Lethality and Rapamycin-Sensitive 5′-Terminal Oligopyrimidine mRNA Translation and Reveal a Mitogen-Activated Protein Kinase-Dependent S6 Kinase Pathway (Molecular and Cellular Biology)
- Translational regulatory mechanisms in persistent forms of synaptic plasticity. (Neuron)
- mTOR interacts with raptor to form a nutrient-sensitive complex that signals to the cell growth machinery. (Cell)
- Target of rapamycin (TOR): an integrator of nutrient and growth factor signals and coordinator of cell growth and cell cycle progression (Oncogene)
- Biallelic Inactivation of BRCA2 in Fanconi Anemia (Science)
- Control of BRCA2 cellular and clinical functions by a nuclear partner, PALB2. (Molecules and Cells)
- Checking on DNA damage in S phase (Nature reviews. Molecular cell biology)
Their papers
- Tumor-promoting phorbol esters and activated Ras inactivate the tuberous sclerosis tumor suppressor complex via p90 ribosomal S6 kinase. (2004) — 797 citations
- ATM and ATR Substrate Analysis Reveals Extensive Protein Networks Responsive to DNA Damage (2007) — 3,199 citations
- mTOR and S6K1 mediate assembly of the translation preinitiation complex through dynamic protein interchange and ordered phosphorylation events. (2005) — 596 citations
- Identification of the FANCI protein, a monoubiquitinated FANCD2 paralog required for DNA repair. (2007) — 739 citations
- Abraxas and RAP80 Form a BRCA1 Protein Complex Required for the DNA Damage Response (2007) — 721 citations
- Probing Transcription Factor Dynamics at the Single-Molecule Level in a Living Cell (2007) — 889 citations