P. Baluk
Molecular Biology · 2000–2007
A researcher in the Science corpus: 6 papers; trusts 2 researchers and is trusted by 4 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 35 citations into it; #9,513 of 17,773 by trust here, #4,951 by citations
- Oncology — 18 citations into it
- Cancer Research — 17 citations into it; #761 of 1,780 by trust here, #665 by citations
Whom P. Baluk trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- Rakesh K. Jain
- H. Hashizume
- Holger Gerhardt
- Raghu Kalluri
- D. McDonald
- Peter Carmeliet
- Lisa M. Coussens
Who trusts P. Baluk overall
- Rakesh K. Jain
- K. Alitalo
- D. Hanahan
- A. Armulik
- Pengfei Lu
- S. Carmichael
- T. Tammela
- G. Bergers
- D. McDonald
- H. Wiig
Papers from P. Baluk's perspective
Ordered by how strongly the researchers P. Baluk trusts endorse each paper. Same corpus, a different order for every researcher.
- Matrix metalloproteinase-9 triggers the angiogenic switch during carcinogenesis (Nature Cell Biology)
- Dissecting tumour pathophysiology using intravital microscopy (Nature Reviews. Cancer)
- Openings between defective endothelial cells explain tumor vessel leakiness. (American Journal of Pathology)
- Normalizing tumor vasculature with anti-angiogenic therapy: A new paradigm for combination therapy (Nature Medicine)
- Abnormalities in pericytes on blood vessels and endothelial sprouts in tumors. (American Journal of Pathology)
- Pathology: Cancer cells compress intratumour vessels (Nature)
- Inhibition of vascular endothelial growth factor (VEGF) signaling in cancer causes loss of endothelial fenestrations, regression of tumor vessels, and appearance of basement membrane ghosts. (American Journal of Pathology)
- Endothelial-pericyte interactions in angiogenesis (Cell and Tissue Research)
- Lack of Pericytes Leads to Endothelial Hyperplasia and Abnormal Vascular Morphogenesis (Journal of Cell Biology)
- Tumor angiogenesis (Advances in Cancer Research)
Their papers
- Inhibition of vascular endothelial growth factor (VEGF) signaling in cancer causes loss of endothelial fenestrations, regression of tumor vessels, and appearance of basement membrane ghosts. (2004) — 759 citations
- Openings between defective endothelial cells explain tumor vessel leakiness. (2000) — 1,654 citations
- Functionally specialized junctions between endothelial cells of lymphatic vessels (2007) — 958 citations
- Cellular abnormalities of blood vessels as targets in cancer. (2005) — 809 citations
- Pathogenesis of persistent lymphatic vessel hyperplasia in chronic airway inflammation. (2005) — 618 citations
- Abnormalities in pericytes on blood vessels and endothelial sprouts in tumors. (2002) — 1,030 citations