Pathogenesis of persistent lymphatic vessel hyperplasia in chronic airway inflammation.

By P. Baluk, T. Tammela, Erin Ator, Natalya Lyubynska, M. Achen, D. Hicklin, M. Jeltsch, T. Petrova, B. Pytowski, S. Stacker and 4 more. Journal of Clinical Investigation, 2005

618 citations recorded by the source corpus.

Subfields: Oncology, Physiology, Surgery

View Paper: https://www.semanticscholar.org/paper/fa8738298ca310313d9f53212402ed0badd38e07

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Oncology

1. H. Wiig
2. K. Alitalo
3. M. Swartz
4. T. Tammela
5. Rakesh K. Jain
6. N. Ferrara
7. M. Shibuya

In Physiology

1. K. Alitalo
2. T. Tammela
3. N. Ferrara

In Surgery

1. K. Alitalo
2. M. Swartz

Citations in the corpus

• Lymphangiogenesis in development and human disease (2005, background): “At least some of the effects of VEGF-A on lymphatic vessels may be secondary to the induction of vascular hyperpermeability and to the recruitment of the inflammatory cells that produce VEGF-C and VEGF-”
• The lymphatic vasculature in disease (2011, background): “Instead of the zipper-like endothelial junctions that occur between BECs, LECs have discontinuous button-like junctions, allowing fluid and certain leukocytes to enter into the vessel lumen through flap-like openings that form the primary lymphatic valve”
• Interstitial fluid and lymph formation and transport: physiological regulation and roles in inflammation and cancer. (2012, background): “In some models of experimental inflammation, VEGFR-3-dependent lymphangiogenesis has been shown to play a role in the resolution of inflammation and edema, including in a mouse model of chronic airway inflammation (34) and inflammation in mouse skin (228, 229).”
• Lymphoid organ development: from ontogeny to neogenesis (2006, background): “Lymphatic vessels have also been noted in murine models of corneal transplantation, suture-induced corneal inflammation and Mycoplasma pulmonis –induced airway inflammatio”
• Lymphangiogenesis: Molecular mechanisms and future promise. (2010, background): “Lymphangiogenesis at sites of inflammatory insults is induced by macrophages and granulocytes, which produce ample amounts of VEGF-C and VEGF-D (Alitalo et al., 2005; Baluk et al., 2005).”