Edward A. Fon

Epidemiology · 2012–2021

A researcher in the Science corpus: 7 papers; trusts 10 researchers and is trusted by 1 researcher here, as inferred from the citation graph.

Subfields

Whom Edward A. Fon trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. Derek P. Narendra
  2. R. Youle
  3. N. Braverman

Who trusts Edward A. Fon overall

  1. I. Slaninová

Papers from Edward A. Fon's perspective

Ordered by how strongly the researchers Edward A. Fon trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Parkin is recruited selectively to impaired mitochondria and promotes their autophagy (Journal of Cell Biology)
  2. PINK1 Is Selectively Stabilized on Impaired Mitochondria to Activate Parkin (PLoS Biology)
  3. PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1 (Nature Cell Biology)
  4. Mitochondrial dysfunction in Drosophila PINK1 mutants is complemented by parkin (Nature)
  5. Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin (Nature)
  6. Mitochondrial pathology and apoptotic muscle degeneration in Drosophila parkin mutants (Proceedings of the National Academy of Sciences of the United States of America)
  7. Fission and selective fusion govern mitochondrial segregation and elimination by autophagy (EMBO Journal)
  8. PINK1 stabilized by mitochondrial depolarization recruits Parkin to damaged mitochondria and activates latent Parkin for mitophagy (Journal of Cell Biology)
  9. Mitochondrial pathology and muscle and dopaminergic neuron degeneration caused by inactivation of Drosophila Pink1 is rescued by Parkin. (Proceedings of the National Academy of Sciences of the United States of America)
  10. The PINK1/Parkin pathway regulates mitochondrial morphology (Proceedings of the National Academy of Sciences of the United States of America)

Their papers