L. Krejci
Molecular Biology · 2003–2012
A researcher in the Science corpus: 2 papers; trusts 2 researchers and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 37 citations into it
- Cancer Research — 5 citations into it
Whom L. Krejci trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts L. Krejci overall
Papers from L. Krejci's perspective
Ordered by how strongly the researchers L. Krejci trusts endorse each paper. Same corpus, a different order for every researcher.
- Differential timing and control of noncrossover and crossover recombination during meiosis. (Cell)
- The Bloom's syndrome helicase suppresses crossing over during homologous recombination (Nature)
- Srs2 and Sgs1-Top3 suppress crossovers during double-strand break repair in yeast. (Cell)
- Targeting the DNA repair defect in BRCA mutant cells as a therapeutic strategy (Nature)
- Control of BRCA2 cellular and clinical functions by a nuclear partner, PALB2. (Molecules and Cells)
- Sensing DNA Damage Through ATRIP Recognition of RPA-ssDNA Complexes (Science)
- The single-end invasion: an asymmetric intermediate at the double-strand break to double-holliday junction transition of meiotic recombination. (Cell)
- DNA helicase Srs2 disrupts the Rad51 presynaptic filament (Nature)
- Mechanism of eukaryotic homologous recombination. (Annual Review of Biochemistry)
- PALB2, which encodes a BRCA2-interacting protein, is a breast cancer susceptibility gene (Nature Genetics)
Their papers
- DNA helicase Srs2 disrupts the Rad51 presynaptic filament (2003) — 632 citations
- Homologous recombination and its regulation (2012) — 661 citations