Ian D. Hickson

Nature · 2001–2011

A researcher in the Science corpus: 7 papers; trusts 0 researchers and is trusted by 17 researchers here, as inferred from the citation graph.

Whom Ian D. Hickson trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. M. Cox

Who trusts Ian D. Hickson overall

1. Manel Esteller
2. P. Sung
3. Leonard Wu
4. L. Krejci
5. Zhu Zhu
6. Xuan Li
7. Eleni P. Mimitou
8. Thomas Helleday
9. Wolf-Dietrich Heyer
10. H. Klein

Papers from Ian D. Hickson's perspective

Ordered by how strongly the researchers Ian D. Hickson trusts endorse each paper. Same corpus, a different order for every researcher.

1. The Bloom's syndrome helicase suppresses crossing over during homologous recombination (Nature)
2. Initiation of genetic recombination and recombination-dependent replication. (TIBS -Trends in Biochemical Sciences. Regular ed)
3. Chromatin relaxation in response to DNA double-strand breaks is modulated by a novel ATM- and KAP-1 dependent pathway (Nature Cell Biology)
4. RNF168 binds and amplifies ubiquitin conjugates on damaged chromosomes to allow accumulation of repair proteins. (Cell)
5. Sister chromatid gene conversion is a prominent double‐strand break repair pathway in mammalian cells (EMBO Journal)
6. 53BP1 loss rescues BRCA1 deficiency and is associated with triple-negative and BRCA-mutated breast cancers (Nature Structural &Molecular Biology)
7. 53BP1 inhibits homologous recombination in Brca1-deficient cells by blocking resection of DNA breaks (Cell)
8. The importance of repairing stalled replication forks (Nature)
9. Differential timing and control of noncrossover and crossover recombination during meiosis. (Cell)
10. The Breast Cancer Susceptibility Gene BRCA1 Is Required for Subnuclear Assembly of Rad51 and Survival following Treatment with the DNA Cross-linking Agent Cisplatin*

Their papers

• Targeting the DNA repair defect in BRCA mutant cells as a therapeutic strategy (2005) — 6,462 citations
• The Bloom's syndrome helicase suppresses crossing over during homologous recombination (2003) — 1,143 citations
• Replication stress induces sister-chromatid bridging at fragile site loci in mitosis (2009) — 600 citations
• RecQ helicases: caretakers of the genome (2003) — 695 citations
• 53BP1 nuclear bodies form around DNA lesions generated by mitotic transmission of chromosomes under replication stress (2011) — 680 citations
• The Bloom's and Werner's syndrome proteins are DNA structure-specific helicases (2001) — 615 citations
• Phosphorylation of BLM, Dissociation from Topoisomerase IIIα, and Colocalization with γ-H2AX after Topoisomerase I-Induced Replication Damage (2005) — 85 citations