F. D. De Felice
Physiology · 2007–2019
A researcher in the Science corpus: 5 papers; trusts 5 researchers and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Physiology — 69 citations into it
- Molecular Biology — 47 citations into it
- Cellular and Molecular Neuroscience — 32 citations into it
- Epidemiology — 23 citations into it
- Pharmacology — 18 citations into it
- Surgery — 12 citations into it
Whom F. D. De Felice trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- Mark P. Mattson
- D. Selkoe
- M. White
- Christian Haass
- K. Petersen
- G. Hotamışlıgil
- K. Viola
- Ana M Fernandez
- V. Samuel
- M. Schwartz
Papers from F. D. De Felice's perspective
Ordered by how strongly the researchers F. D. De Felice trusts endorse each paper. Same corpus, a different order for every researcher.
- Pathways towards and away from Alzheimer's disease (Nature)
- Naturally secreted oligomers of amyloid β protein potently inhibit hippocampal long-term potentiation in vivo (Nature)
- Mechanism by which high-dose aspirin improves glucose metabolism in type 2 diabetes. (Journal of Clinical Investigation)
- Defects in IGF-1 receptor, insulin receptor and IRS-1/2 in Alzheimer's disease indicate possible resistance to IGF-1 and insulin signalling. (Neurobiology of Aging)
- Phosphorylation of Ser307 in Insulin Receptor Substrate-1 Blocks Interactions with the Insulin Receptor and Inhibits Insulin Action* (Journal of Biological Chemistry)
- Regulation of NMDA receptor trafficking by amyloid-β (Nature Neuroscience)
- Intranasal insulin improves cognition and modulates β-amyloid in early AD (Neurology)
- Chronic inflammation in fat plays a crucial role in the development of obesity-related insulin resistance. (Journal of Clinical Investigation)
- Increased risk of type 2 diabetes in Alzheimer disease. (Diabetes)
- Soluble oligomers of the amyloid beta-protein impair synaptic plasticity and behavior. (Behavioural Brain Research)
Their papers
- Exercise-linked FNDC5/irisin rescues synaptic plasticity and memory defects in Alzheimer’s models (2019) — 801 citations
- Inflammation, Defective Insulin Signaling, and Mitochondrial Dysfunction as Common Molecular Denominators Connecting Type 2 Diabetes to Alzheimer Disease (2014) — 606 citations
- An anti-diabetes agent protects the mouse brain from defective insulin signaling caused by Alzheimer's disease- associated Aβ oligomers. (2012) — 813 citations
- Protection of synapses against Alzheimer's-linked toxins: Insulin signaling prevents the pathogenic binding of Aβ oligomers (2009) — 616 citations
- Aβ Oligomers Induce Neuronal Oxidative Stress through an N-Methyl-D-aspartate Receptor-dependent Mechanism That Is Blocked by the Alzheimer Drug Memantine* (2007) — 904 citations