Hiroshi Takayanagi

Molecular Biology · 2000–2019

A researcher in the Science corpus: 12 papers; trusts 15 researchers and is trusted by 4 researchers here, as inferred from the citation graph.

Subfields

• Molecular Biology — 91 citations into it; #10,024 of 17,773 by trust here, #3,204 by citations
• Immunology — 66 citations into it; #1,234 of 2,160 by trust here, #865 by citations
• Oncology — 66 citations into it; #1,492 of 1,698 by trust here, #620 by citations
• Orthopedics and Sports Medicine — 23 citations into it
• Cancer Research — 20 citations into it; #1,745 of 1,780 by trust here, #586 by citations
• Genetics — 20 citations into it
• Hematology — 20 citations into it
• Rheumatology — 14 citations into it

Whom Hiroshi Takayanagi trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. W. J. Boyle
2. Shizuo Akira
3. N. Nishimoto
4. S. Teitelbaum
5. P. Hogan
6. G. Karsenty
7. G. Rodan
8. S. Manolagas
9. Zhijian J. Chen
10. E. Wagner

Who trusts Hiroshi Takayanagi overall

1. W. J. Boyle
2. B. Boyce
3. S. Teitelbaum
4. S. Gordon
5. W. S. Simonet
6. R. Florêncio-Silva
7. Georg Schett
8. Xiaoyu Hu
9. L. Xing
10. J. H. Park

Papers from Hiroshi Takayanagi's perspective

Ordered by how strongly the researchers Hiroshi Takayanagi trusts endorse each paper. Same corpus, a different order for every researcher.

1. Therapeutic approaches to bone diseases. (Science)
2. T-cell-mediated regulation of osteoclastogenesis by signalling cross-talk between RANKL and IFN-γ (Nature)
3. Genetic regulation of osteoclast development and function (Nature reviews genetics)
4. Birth and death of bone cells: basic regulatory mechanisms and implications for the pathogenesis and treatment of osteoporosis. (Endocrine reviews)
5. RANKL maintains bone homeostasis through c-Fos-dependent induction of interferon-β (Nature)
6. IRF family of transcription factors as regulators of host defense. (Annual Review of Immunology)
7. Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells (Nature)
8. TRANCE/RANKL knockout mice are protected from bone erosion in a serum transfer model of arthritis. (American Journal of Pathology)
9. Natural killer cells, viruses and cancer (Nature reviews. Immunology)
10. Transforming growth factor-beta induces development of the T(H)17 lineage. (Nature)

Their papers

• Induction and activation of the transcription factor NFATc1 (NFAT2) integrate RANKL signaling in terminal differentiation of osteoclasts. (2002) — 2,488 citations
• Th17 functions as an osteoclastogenic helper T cell subset that links T cell activation and bone destruction (2006) — 1,580 citations
• T-cell-mediated regulation of osteoclastogenesis by signalling cross-talk between RANKL and IFN-γ (2000) — 1,093 citations
• Costimulatory signals mediated by the ITAM motif cooperate with RANKL for bone homeostasis (2004) — 855 citations
• Evidence for osteocyte regulation of bone homeostasis through RANKL expression (2011) — 1,711 citations
• RANKL maintains bone homeostasis through c-Fos-dependent induction of interferon-β (2002) — 761 citations
• Osteoimmunology: shared mechanisms and crosstalk between the immune and bone systems (2007) — 1,823 citations
• The molecular understanding of osteoclast differentiation. (2007) — 1,333 citations
• Estrogen Prevents Bone Loss via Estrogen Receptor α and Induction of Fas Ligand in Osteoclasts (2007) — 1,011 citations
• Osteoimmunology: evolving concepts in bone–immune interactions in health and disease (2019) — 648 citations
• Pathogenic conversion of Foxp3+ T cells into TH17 cells in autoimmune arthritis (2013) — 1,087 citations
• Involvement of receptor activator of nuclear factor kappaB ligand/osteoclast differentiation factor in osteoclastogenesis from synoviocytes in rheumatoid arthritis. (2000) — 635 citations