Estrogen Prevents Bone Loss via Estrogen Receptor α and Induction of Fas Ligand in Osteoclasts
By Takashi Nakamura, Yuuki Imai, Takahiro Matsumoto, Shingo Sato, Kazusane Takeuchi, K. Igarashi, Y. Harada, Y. Azuma, A. Krust, Yoko Yamamoto and 9 more. 2007
1,011 citations recorded by the source corpus.
Subfields: Genetics, Molecular Biology, Oncology
View Paper: https://www.semanticscholar.org/paper/ed55d686f6ffc6ce14c3aab58bf5684ab8a6a376
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Citations in the corpus
- Estrogen and the Skeleton (2012, neutral): “These findings contrast with the clear phenotype noted earlier in mice with osteoclastspecific deletion of ERα (28, 29).”
- Disorders of bone remodeling. (2011, background): “Second, estrogen directly targets mature osteoclasts to promote apoptosis of osteoclasts via the action of the Fas/Fas ligand system (75, 76).”
- Endocrine regulation of male fertility by the skeleton. (2011, neutral): “To date, the study of the interplay between gonads and bone has focused on the mechanism whereby sex steroid hormones affect bone mass accrual (Manolagas et al., 2002; Nakamura et al., 2007).”