David H. Gross
Molecular Biology · 2008
A researcher in the Science corpus: 1 paper; trusts 3 researchers and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 36 citations into it
- Physiology — 20 citations into it
- Epidemiology — 9 citations into it
Whom David H. Gross trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Papers from David H. Gross's perspective
Ordered by how strongly the researchers David H. Gross trusts endorse each paper. Same corpus, a different order for every researcher.
- Foxo transcription factors induce the atrophy-related ubiquitin ligase atrogin-1 and cause skeletal muscle atrophy. (Cell)
- The IGF-1/PI3K/Akt pathway prevents expression of muscle atrophy-induced ubiquitin ligases by inhibiting FOXO transcription factors. (Molecules and Cells)
- Regulation of insulin action and pancreatic β-cell function by mutated alleles of the gene encoding forkhead transcription factor Foxo1 (Nature Genetics)
- Atrogin-1, a muscle-specific F-box protein highly expressed during muscle atrophy (Proceedings of the National Academy of Sciences of the United States of America)
- Sirt1 promotes fat mobilization in white adipocytes by repressing PPAR-γ (Nature)
- Insulin-regulated hepatic gluconeogenesis through FOXO1–PGC-1α interaction (Nature)
- Identification of Ubiquitin Ligases Required for Skeletal Muscle Atrophy (Science)
- The forkhead transcription factor Foxo1 regulates adipocyte differentiation. (Developmental Cell)
- FoxOs at the crossroads of cellular metabolism, differentiation, and transformation. (Cell)
- Increased dosage of a sir-2 gene extends lifespan in Caenorhabditis elegans (Nature)
Their papers
- The role of FoxO in the regulation of metabolism (2008) — 647 citations