K. Moore

Molecular Biology · Nature Immunology · 2002–2023

A researcher in the Science corpus: 12 papers; trusts 7 researchers and is trusted by 4 researchers here, as inferred from the citation graph.

Subfields

• Molecular Biology — 173 citations into it
• Immunology — 66 citations into it; #550 of 2,160 by trust here, #280 by citations
• Surgery — 42 citations into it; #1,087 of 1,106 by trust here, #123 by citations
• Epidemiology — 26 citations into it; #518 of 2,148 by trust here, #858 by citations
• Cell Biology — 5 citations into it
• Endocrinology — 5 citations into it

Whom K. Moore trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. I. Tabas
2. Christopher K. Glass
3. Fabio Martinon
4. J. Witztum
5. F. Tacke
6. X. Chen
7. L. Stuart
8. F. Maxfield
9. Ann-Hwee Lee

Who trusts K. Moore overall

1. K. Bornfeldt
2. Christopher K. Glass
3. I. Tabas
4. Jerrold M. Olefsky
5. P. Murray
6. Sophie Colin
7. J. Bentzon
8. T. Postler
9. G. Chinetti-Gbaguidi

Papers from K. Moore's perspective

Ordered by how strongly the researchers K. Moore trusts endorse each paper. Same corpus, a different order for every researcher.

1. Cryopyrin activates the inflammasome in response to toxins and ATP (Nature)
2. Gout-associated uric acid crystals activate the NALP3 inflammasome (Nature)
3. Toll-like receptor signalling (Nature reviews. Immunology)
4. Toll-like receptor 4 polymorphisms and atherogenesis. (New England Journal of Medicine)
5. Cutting Edge: NF-κB Activating Pattern Recognition and Cytokine Receptors License NLRP3 Inflammasome Activation by Regulating NLRP3 Expression1 (Journal of Immunology)
6. The repertoire for pattern recognition of pathogens by the innate immune system is defined by cooperation between toll-like receptors. (Proceedings of the National Academy of Sciences of the United States of America)
7. Lack of Interleukin-1&bgr; Decreases the Severity of Atherosclerosis in ApoE-Deficient Mice (Arteriosclerosis, Thrombosis and Vascular Biology)
8. CD36 is a sensor of diacylglycerides (Nature)
9. The NALP3 inflammasome is involved in the innate immune response to amyloid-β (Nature Immunology)
10. Scavenger Receptors Class A-I/II and CD36 Are the Principal Receptors Responsible for the Uptake of Modified Low Density Lipoprotein Leading to Lipid Loading in Macrophages* (Journal of Biological Chemistry)

Their papers

• The NALP3 inflammasome is involved in the innate immune response to amyloid-β (2008) — 1,957 citations
• MiR-33 Contributes to the Regulation of Cholesterol Homeostasis (2010) — 1,302 citations
• NLRP3 inflamasomes are required for atherogenesis and activated by cholesterol crystals that form early in disease (2010) — 2,600 citations
• Long non-coding RNAs: definitions, functions, challenges and recommendations (2023) — 1,816 citations
• Scavenger Receptors Class A-I/II and CD36 Are the Principal Receptors Responsible for the Uptake of Modified Low Density Lipoprotein Leading to Lipid Loading in Macrophages* (2002) — 1,046 citations
• CD36 coordinates NLRP3 inflammasome activation by facilitating the intracellular nucleation from soluble to particulate ligands in sterile inflammation (2013) — 619 citations
• Antagonism of miR-33 in mice promotes reverse cholesterol transport and regression of atherosclerosis. (2011) — 725 citations
• Macrophages in atherosclerosis: a dynamic balance (2013) — 2,448 citations
• The Cellular Biology of Macrophages in Atherosclerosis (2011) — 2,598 citations
• miR-33a/b contribute to the regulation of fatty acid metabolism and insulin signaling (2011) — 677 citations
• CD36 ligands promote sterile inflammation through assembly of a Toll-like receptor 4 and 6 heterodimer (2009) — 1,527 citations
• Reduced atherosclerosis in MyD88-null mice links elevated serum cholesterol levels to activation of innate immunity signaling pathways (2004) — 693 citations