J. Hayes

Molecular Biology · TIBS -Trends in Biochemical Sciences. Regular ed · 2000–2020

A researcher in the Science corpus: 13 papers; trusts 22 researchers and is trusted by 13 researchers here, as inferred from the citation graph.

Subfields

Molecular Biology — 275 citations into it; #3,214 of 17,773 by trust here, #2,180 by citations
Epidemiology — 37 citations into it
Cell Biology — 36 citations into it
Cancer Research — 34 citations into it
Oncology — 34 citations into it
Immunology — 32 citations into it
Physiology — 24 citations into it; #1,924 of 2,276 by trust here, #1,591 by citations
Nutrition and Dietetics — 23 citations into it

Whom J. Hayes trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

Who trusts J. Hayes overall

Papers from J. Hayes's perspective

Ordered by how strongly the researchers J. Hayes trusts endorse each paper. Same corpus, a different order for every researcher.

Cell survival responses to environmental stresses via the Keap1-Nrf2-ARE pathway. (Annual Review of Pharmacology and Toxicology)
Oxidative Stress Sensor Keap1 Functions as an Adaptor for Cul3-Based E3 Ligase To Regulate Proteasomal Degradation of Nrf2 (Molecular and Cellular Biology)
Oncogene-induced Nrf2 transcription promotes ROS detoxification and tumorigenesis (Nature)
Cancer related mutations in NRF2 impair its recognition by Keap1-Cul3 E3 ligase and promote malignancy (Proceedings of the National Academy of Sciences of the United States of America)
Nrf2 Is a Direct PERK Substrate and Effector of PERK-Dependent Cell Survival (Molecular and Cellular Biology)
Nrf2-Keap1 regulation of cellular defense mechanisms against electrophiles and reactive oxygen species. (Advances in Enzyme Regulation)
Regulation of Ferroptotic Cancer Cell Death by GPX4 (Cell)
Ferroptosis: an iron-dependent form of nonapoptotic cell death. (Cell)
Peroxidation of polyunsaturated fatty acids by lipoxygenases drives ferroptosis (Proceedings of the National Academy of Sciences of the United States of America)
How mitochondria produce reactive oxygen species (Biochemical Journal)

Their papers

p62/SQSTM1 Is a Target Gene for Transcription Factor NRF2 and Creates a Positive Feedback Loop by Inducing Antioxidant Response Element-driven Gene Transcription* (2010) — 1,400 citations
Keap1-dependent Proteasomal Degradation of Transcription Factor Nrf2 Contributes to the Negative Regulation of Antioxidant Response Element-driven Gene Expression* (2003) — 1,150 citations
NRF2 and KEAP1 mutations: permanent activation of an adaptive response in cancer. (2009) — 869 citations
SCF/β-TrCP Promotes Glycogen Synthase Kinase 3-Dependent Degradation of the Nrf2 Transcription Factor in a Keap1-Independent Manner (2011) — 729 citations
Nrf2 is controlled by two distinct β-TrCP recognition motifs in its Neh6 domain, one of which can be modulated by GSK-3 activity. (2012) — 642 citations
The Nrf2 regulatory network provides an interface between redox and intermediary metabolism. (2014) — 2,005 citations
Glutathione transferases. (2005) — 1,339 citations
Glutathione S-Transferase Polymorphisms and Their Biological Consequences (2000) — 1,016 citations
Mechanisms of activation of the transcription factor Nrf2 by redox stressors, nutrient cues, and energy status and the pathways through which it attenuates degenerative disease (2015) — 796 citations
Therapeutic targeting of the NRF2 and KEAP1 partnership in chronic diseases (2019) — 1,270 citations
Oxidative Stress in Cancer (2020) — 1,890 citations
Glutathione S‐transferases (2002) — 1,383 citations
The Cap'n'Collar basic leucine zipper transcription factor Nrf2 (NF-E2 p45-related factor 2) controls both constitutive and inducible expression of intestinal detoxification and glutathione biosynthetic enzymes. (2001) — 701 citations