Pramod Kumar Srivastava

Molecular Biology · 2000–2002

A researcher in the Science corpus: 3 papers; trusts 1 researcher and is trusted by 15 researchers here, as inferred from the citation graph.

Subfields

Whom Pramod Kumar Srivastava trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. T. Ramalingam

Who trusts Pramod Kumar Srivastava overall

  1. Bali Pulendran
  2. Rafi Ahmed
  3. O. Finn
  4. T. Waldmann
  5. C. Théry
  6. D. Ciocca
  7. R. Lake
  8. G. Dunn
  9. Stuart K. Calderwood
  10. Y. Sung

Papers from Pramod Kumar Srivastava's perspective

Ordered by how strongly the researchers Pramod Kumar Srivastava trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Consequences of Cell Death (Journal of Experimental Medicine)
  2. Selective Suppression of Endothelial Cell Apoptosis by the High Molecular Weight Form of Adiponectin (Circulation Research)
  3. Disruption of Adiponectin Causes Insulin Resistance and Neointimal Formation* (Journal of Biological Chemistry)
  4. Proteolytic cleavage product of 30-kDa adipocyte complement-related protein increases fatty acid oxidation in muscle and causes weight loss in mice. (Proceedings of the National Academy of Sciences of the United States of America)
  5. Plasma concentrations of a novel, adipose-specific protein, adiponectin, in type 2 diabetic patients. (Arteriosclerosis, Thrombosis and Vascular Biology)
  6. Akt promotes survival of cardiomyocytes in vitro and protects against ischemia-reperfusion injury in mouse heart. (Circulation)
  7. Reciprocal Association of C-Reactive Protein With Adiponectin in Blood Stream and Adipose Tissue (Circulation)
  8. Adiponectin Stimulates Angiogenesis by Promoting Cross-talk between AMP-activated Protein Kinase and Akt Signaling in Endothelial Cells* (Journal of Biological Chemistry)
  9. The inflammatory response in myocardial infarction. (Cardiovascular Research)
  10. AMP-activated protein kinase mediates ischemic glucose uptake and prevents postischemic cardiac dysfunction, apoptosis, and injury. (Journal of Clinical Investigation)

Their papers