The inflammatory response in myocardial infarction.

By Nikolaos G. Frangogiannis, C. Smith, M. Entman. Cardiovascular Research, 2002

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Protective Actions of H2S in Acute Myocardial Infarction and Heart Failure (2017, neutral): “resulting in the loss of viable myocardial tissue (47).”
Adiponectin protects against myocardial ischemia-reperfusion injury through AMPK- and COX-2–dependent mechanisms (2005, background): “Elevated production of TNF-α in APN-KO mice Because inflammation contributes to myocardial injury after ischemia and reperfusio”
The IL-33/ST2 pathway: therapeutic target and novel biomarker (2008, background): “In animal models, manipulation of this inflammatory-cell infiltration results in attenuation of the severity of experimental infarction (reviewed in Ref”
Involvement of neutrophils in the pathogenesis of lethal myocardial reperfusion injury. (2004, background): “In addition, recent evidence suggests that cytokines such as TNFa can be released by degranulation of mast cells during ischemia–reperfusion [33,34].”
Cell Biology of Ischemia/Reperfusion Injury (2012, background): “Subsequent studies demonstrated cell necrosis induced by ischemia results in release of subcellular membrane constituents that trigger the complement cascade (Frangogiannis et al., 2002; Ioannou et al., 2011; Rossen et al., 1994).”