F. Quintana
Immunology · Nature · 2008–2021
A researcher in the Science corpus: 17 papers; trusts 9 researchers and is trusted by 6 researchers here, as inferred from the citation graph.
Subfields
- Immunology — 222 citations into it; #588 of 2,160 by trust here, #666 by citations
- Neurology — 101 citations into it
- Physiology — 54 citations into it
- Cellular and Molecular Neuroscience — 43 citations into it
- Biological Psychiatry — 41 citations into it
- Developmental Neuroscience — 32 citations into it
- Cancer Research — 17 citations into it
- Health, Toxicology and Mutagenesis — 12 citations into it
Whom F. Quintana trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- T. Korn
- E. Bettelli
- M. Sofroniew
- N. J. Allen
- M. Veldhoen
- D. Mucida
- Christopher A. Bradfield
- J. Fontenot
Who trusts F. Quintana overall
Papers from F. Quintana's perspective
Ordered by how strongly the researchers F. Quintana trusts endorse each paper. Same corpus, a different order for every researcher.
- Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells (Nature)
- Regulatory T cell lineage specification by the forkhead transcription factor foxp3. (Immunity)
- Interleukin 27 negatively regulates the development of interleukin 17–producing T helper cells during chronic inflammation of the central nervous system (Nature Immunology)
- Interleukin 27 limits autoimmune encephalomyelitis by suppressing the development of interleukin 17–producing T cells (Nature Immunology)
- Reciprocal TH17 and Regulatory T Cell Differentiation Mediated by Retinoic Acid (Science)
- TGF-β and IL-6 drive the production of IL-17 and IL-10 by T cells and restrain TH-17 cell–mediated pathology (Nature Immunology)
- Lamina propria macrophages and dendritic cells differentially induce regulatory and interleukin 17–producing T cell responses (Nature Immunology)
- Essential autocrine regulation by IL-21 in the generation of inflammatory T cells (Nature)
- Myelin-specific regulatory T cells accumulate in the CNS but fail to control autoimmune inflammation (Nature Medicine)
- Interleukin-17-producing gammadelta T cells selectively expand in response to pathogen products and environmental signals. (Immunity)
Their papers
- Alterations of the human gut microbiome in multiple sclerosis (2016) — 1,171 citations
- The aryl hydrocarbon receptor: an environmental sensor integrating immune responses in health and disease (2019) — 1,005 citations
- Tryptophan metabolism drives dynamic immunosuppressive myeloid states in IDH-mutant gliomas (2021) — 196 citations
- Reactive astrocyte nomenclature, definitions, and future directions (2021) — 1,941 citations
- Negative feedback control of neuronal activity by microglia (2020) — 814 citations
- Astrocyte crosstalk in CNS inflammation (2020) — 851 citations
- Regulation of the immune response by the aryl hydrocarbon receptor (2018) — 857 citations
- Microglial control of astrocytes in response to microbial metabolites (2018) — 899 citations
- Tolerogenic dendritic cells (2016) — 647 citations
- Type I interferons and microbial metabolites of tryptophan modulate astrocyte activity and CNS inflammation via the aryl hydrocarbon receptor (2016) — 1,114 citations
- [Immunopathology of multiple sclerosis]. (2014) — 635 citations
- Treg cells expressing the co-inhibitory molecule TIGIT selectively inhibit pro-inflammatory Th1 and Th17 cell responses (2014) — 877 citations
- IL-21 induces IL-22 production in CD4+ T-cells (2014) — 148 citations
- Human polymorphism at microRNAs and microRNA target sites. (2013) — 605 citations
- Induction and molecular signature of pathogenic TH17 cells (2012) — 1,152 citations
- Oral tolerance (2011) — 672 citations
- Control of Treg and TH17 cell differentiation by the aryl hydrocarbon receptor (2008) — 1,239 citations