A. Steinle
Immunology · 2008
A researcher in the Science corpus: 1 paper; trusts 7 researchers and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Immunology — 28 citations into it
- Oncology — 12 citations into it
Whom A. Steinle trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Papers from A. Steinle's perspective
Ordered by how strongly the researchers A. Steinle trusts endorse each paper. Same corpus, a different order for every researcher.
- ULBPs, novel MHC class I-related molecules, bind to CMV glycoprotein UL16 and stimulate NK cytotoxicity through the NKG2D receptor. (Immunity)
- Retinoic acid early inducible genes define a ligand family for the activating NKG2D receptor in mice. (Immunity)
- Licensing of natural killer cells by host major histocompatibility complex class I molecules (Nature)
- Activating receptors and coreceptors involved in human natural killer cell-mediated cytolysis. (Annual Review of Immunology)
- IFNγ and lymphocytes prevent primary tumour development and shape tumour immunogenicity (Nature)
- Tumour-derived soluble MIC ligands impair expression of NKG2D and T-cell activation (Nature)
- Rae1 and H60 ligands of the NKG2D receptor stimulate tumour immunity (Nature)
- Roles of the NKG2D immunoreceptor and its ligands (Nature reviews. Immunology)
- NK cell recognition. (Annual Review of Immunology)
- Ectopic expression of retinoic acid early inducible-1 gene (RAE-1) permits natural killer cell-mediated rejection of a MHC class I-bearing tumor in vivo (Proceedings of the National Academy of Sciences of the United States of America)
Their papers
- NK cells and cancer immunosurveillance (2008) — 675 citations