Physiological dysfunction of the dorsolateral prefrontal cortex in schizophrenia revisited.
By J. Callicott, A. Bertolino, Venkata S. Mattay, Frederick J. P. Langheim, Jeff H. Duyn, Richard Coppola, Terry E. Goldberg, Daniel R. Weinberger. Cerebral Cortex, 2000
860 citations recorded by the source corpus.
Subfields: Cognitive Neuroscience, Psychiatry and Mental health, Radiology, Nuclear Medicine and Imaging
View Paper: https://www.semanticscholar.org/paper/eac5431ac73e4652da001db8c11bbc40916599de
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Cognitive Neuroscience
- J. Cohen
- Cameron S. Carter
- Bharat B. Biswal
- Mark D'Esposito
- Angela R. Laird
- Matthew Botvinick
- Michael F. Green
- Daniel R. Weinberger
In Psychiatry and Mental health
- Daniel R. Weinberger
- Michael F. Green
- Dara S. Manoach
- R. Heaton
- Deanna M. Barch
- Venkata S. Mattay
- Bhaskar S. Kolachana
- Anissa Abi-Dargham
In Radiology, Nuclear Medicine and Imaging
- Mark D'Esposito
- Daniel R. Weinberger
- J. Cohen
- Arno Villringer
- Marc Laruelle
- Daniel Y. Kimberg
- Georg Winterer
- Bhaskar S. Kolachana
Citations in the corpus
- Meta-analysis of 41 Functional Neuroimaging Studies of Executive Function in Schizophrenia (2009, background): “Forty-one studies published prior to February 2007 met these criteria (Table 1).(3,5,13-51) Study design features are indicated in Table 1, including clinical characteristics like illness duration and symptom severity; sex ratio among the patient group; medication status; block vs event-related desi”
- Complexity of prefrontal cortical dysfunction in schizophrenia: more than up or down. (2003, background): “First, patients and healthy subjects might operate on the same proximal limb of the loadresponse curve, but patients might reach their capacity sooner (consistent with their limited working memory capacity [3, 4]), fall off the curve sooner, and thus appear to be hypofrontal compared with healthy su”
- Prefrontal Dopamine D1 Receptors and Working Memory in Schizophrenia (2002, background): “…by numerous investigators, suggesting that pathology of the DLPFC or its connectivity is implicated in working memory deficits in schizophrenia (Callicott et al., 1998, 2000; Stevens et al., 1998; Honey et al., 1999; Manoach et al., 1999, 2000; Barch et al., 2001; Perlstein et al., 2001).”
- Neural correlates of cognitive efficiency (2006, methodology): “One study, for instance (Callicott et al., 2000), compared PFC activity of high-functioning schizophrenics and controls during performance of a continuous performance task (CPT) in which, on each trial, 4-item arrays of digits appeared and subjects were required to match them with those seen a presp”
- Dysfunction of ventral striatal reward prediction in schizophrenia (2006, background): “…has long focused on the neurobiological correlates of cognitive dysfunction in prefrontal – striatal – thalamic neurocircuits (Andreasen et al., 1996; Callicott et al., 2000; Meyer-Lindenberg et al., 2002, 2005; Abi-Dargham et al., 2002; Molina et al., 2003), only a few imaging studies have…”
- Prefrontal cortex dysfunction during working memory performance in schizophrenia: reconciling discrepant findings. (2003, background): “Several studies reported increased DLPFC activation when schizophrenic subjects performed above chance but worse than healthy subjects (Callicott et al., 2000; Manoach et al., 1999, 2000).”
- Relation of prefrontal cortex dysfunction to working memory and symptoms in schizophrenia. (2001, background): “These results complement previous findings of dorsolateral prefrontal cortex dysfunction using the n-back task (4, 31, 41).”
- Beyond hypofrontality: A quantitative meta‐analysis of functional neuroimaging studies of working memory in schizophrenia (2005, background): “, 1996], more recent studies have stressed that DLPFC activation in patients is linked to performance deficits, with good performance associated with relatively greater DLPFC activity and marked behavioral deficits linked to relatively reduced DLPFC signal [Callicott et al., 2000; Perlstein et al., ”
- Genes, dopamine and cortical signal-to-noise ratio in schizophrenia. (2004, background): “compared with normal controls when performance on the task does not differ between comparisons groups [ 26 ,64].”
- The BDNF val66met polymorphism affects activity-dependent secretion of BDNF and human memory and hippocampal function. (2003, methodology): “…subjects must recall a number [1, 2, 3, or 4] seen two stimuli from one ethnic group (the largest, European Americans); the latter previously) and a control condition (“0B”; subjects identified the are reported only if different from the former. number currently seen) (Callicott et al., 2000).”