Relation of prefrontal cortex dysfunction to working memory and symptoms in schizophrenia.

By W. Perlstein, C. Carter, D. Noll, J. Cohen. American Journal of Psychiatry, 2001

659 citations recorded by the source corpus.

Subfields: Cognitive Neuroscience, Psychiatry and Mental health

View Paper: https://www.semanticscholar.org/paper/18c884f2a125e3b8f04ec7c5451c3815ad203182

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In Cognitive Neuroscience

In Psychiatry and Mental health

Citations in the corpus

A Research Agenda For DSM-V (2002, methodology): “Because of its temporal and spatial resolution, lack of radiation, and availability, fMRI has become the major tool for this research methodology (Mitchell et al. 2001; Perlstein et al. 2001).”
Prefrontal cortex dysfunction during working memory performance in schizophrenia: reconciling discrepant findings. (2003, background): “Three other recent studies have reported equivalent prefrontal activation with matched performance using the n-back task, two by comparing across WM loads (Callicott et al., 2000; Perlstein et al., 2001) and one by comparing groups at a low WM load (Honey et al.”
Prefrontal Dopamine D1 Receptors and Working Memory in Schizophrenia (2002, background): “Alterations in DLPFC activation during completion of working memory tasks has been reported in patients with schizophrenia by numerous investigators, suggesting that pathology of the DLPFC or its connectivity is implicated in working memory deficits in schizophrenia (Callicott et al., 1998, 2000; St”
Complexity of prefrontal cortical dysfunction in schizophrenia: more than up or down. (2003, neutral): “[15]; and d=1.”
Cortical inhibitory neurons and schizophrenia (2005, background): “Individuals with schizophrenia tend to perform poorly on working memory tasks and to show reduced DLPFC activation when attempting to carry out such task”
Beyond hypofrontality: A quantitative meta‐analysis of functional neuroimaging studies of working memory in schizophrenia (2005, background): “, 1996], more recent studies have stressed that DLPFC activation in patients is linked to performance deficits, with good performance associated with relatively greater DLPFC activity and marked behavioral deficits linked to relatively reduced DLPFC signal [Callicott et al., 2000; Perlstein et al., ”