Abdominal obesity and metabolic syndrome

By J. Despres, I. Lemieux. Nature, 2006

4,593 citations recorded by the source corpus.

Subfields: Cardiology and Cardiovascular Medicine, Endocrinology, Diabetes and Metabolism, Epidemiology

View Paper: https://www.semanticscholar.org/paper/12903a24428f6c6024aed0c7b8de08306f8b15ff

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cardiology and Cardiovascular Medicine

1. André Tchernof
2. M. Blüher
3. J. Despres
4. E. Dale Abel
5. D. J. Drucker
6. Herbert Tilg
7. B. Spiegelman
8. E. Rosen

In Endocrinology, Diabetes and Metabolism

1. André Tchernof
2. M. Blüher
3. S. Klein
4. J. Despres
5. A. Okunade
6. B. Patterson
7. D. J. Drucker
8. Herbert Tilg

In Epidemiology

1. André Tchernof
2. S. Klein
3. A. Okunade
4. B. Patterson
5. S. Fried
6. J. Despres
7. Johan Auwerx
8. L. Guarente

Citations in the corpus

• Metabolic Syndrome and Insulin Resistance: Underlying Causes and Modification by Exercise Training (2013, neutral): “Despres JP, Lemieux I. Abdominal obesity and metabolic syndrome.”
• Intrahepatic fat, not visceral fat, is linked with metabolic complications of obesity (2009, background): “The results of the present study contradict the prevailing dogma that VAT has deleterious metabolic effects (5, 6).”
• Effects of targeted delivery of propionate to the human colon on appetite regulation, body weight maintenance and adiposity in overweight adults (2014, result): “44 Interestingly, in this long-term study we were unable to detect any change in PYY or GLP-1 release following ingestion of the inulin-propionate ester compared with inulin-control, in contrast to our acute administration studies.”
• Cardiac remodeling in obesity. (2008, background): “There is ever-increasing interest in the link between obesity and inflammation (78, 80, 204, 221), and an inflammatory state is both a causative factor in inducing MI and a primary constituent of the response to MI (210, 341, 385).”
• Dietary factors and low-grade inflammation in relation to overweight and obesity (2011, neutral): “Using the data from NHANES III on a representative sample of the US population, Ford et al.(300) found a negative correlation between the HEI and CRP concentration after adjustment for several CVD risk factors including BMI and waist:hip ratio; when stratified by sex this finding was signifi- cant i”
• ESPEN guidelines on definitions and terminology of clinical nutrition. (2017, background): “The presence of central obesity (also known as abdominal, visceral, upper-body or android obesity) can be clinically defined by increased waist circumference (WC) measured in the mid-horizontal plane between the superior iliac crest and the lower margin of the last rib [36].”
• Morbidity and mortality associated with obesity. (2017, background): “factors which appear more useful than the use of BMI alone (6,7).”
• Adipose Tissue Heterogeneity: Implication of depot differences in adipose tissue for Obesity Complications (2012, background): “Sex differences in metabolic risk are at least partially due to the higher visceral fat mass in men (Despres and Lemieux, 2006).”