Lucas B. Sullivan

Molecular Biology · 2011–2016

A researcher in the Science corpus: 5 papers; trusts 9 researchers and is trusted by 4 researchers here, as inferred from the citation graph.

Subfields

Whom Lucas B. Sullivan trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. G. Semenza
  2. M. Brand
  3. Toren Finkel
  4. Lewis C. Cantley
  5. M. Murphy
  6. Laura A. Sena
  7. Donna D. Zhang
  8. R. Hamanaka
  9. K. Conklin
  10. Navdeep S. Chandel

Who trusts Lucas B. Sullivan overall

  1. M. V. Vander Heiden
  2. Ralph J. Deberardinis
  3. W. Zong
  4. E. Panieri
  5. Joshua D. Rabinowitz
  6. C. Lyssiotis
  7. N. Pavlova

Papers from Lucas B. Sullivan's perspective

Ordered by how strongly the researchers Lucas B. Sullivan trusts endorse each paper. Same corpus, a different order for every researcher.

  1. c-Myc suppression of miR-23 enhances mitochondrial glutaminase and glutamine metabolism (Nature)
  2. Beyond aerobic glycolysis: Transformed cells can engage in glutamine metabolism that exceeds the requirement for protein and nucleotide synthesis (Proceedings of the National Academy of Sciences of the United States of America)
  3. Oxygen sensing requires mitochondrial ROS but not oxidative phosphorylation. (Cell Metabolism)
  4. Voltage-dependent Anion Channels Control the Release of the Superoxide Anion from Mitochondria to Cytosol* (Journal of Biological Chemistry)
  5. Hypoxia-Inducible Factors in Physiology and Medicine (Cell)
  6. Why do cancers have high aerobic glycolysis? (Nature Reviews. Cancer)
  7. Mitochondrial metabolism and ROS generation are essential for Kras-mediated tumorigenicity (Proceedings of the National Academy of Sciences of the United States of America)
  8. Analysis of lung tumor initiation and progression using conditional expression of oncogenic K-ras. (Genes & Development)
  9. Oncogene-induced Nrf2 transcription promotes ROS detoxification and tumorigenesis (Nature)
  10. Tumor suppressors and cell metabolism: a recipe for cancer growth. (Genes & Development)

Their papers