M. Calkins

Molecular Biology · Human Molecular Genetics · 2003–2011

A researcher in the Science corpus: 4 papers; trusts 3 researchers and is trusted by 3 researchers here, as inferred from the citation graph.

Subfields

• Molecular Biology — 47 citations into it; #12,161 of 17,773 by trust here, #11,410 by citations
• Physiology — 45 citations into it; #1,223 of 2,276 by trust here, #1,627 by citations
• Pharmacology — 24 citations into it
• Computational Theory and Mathematics — 6 citations into it

Whom M. Calkins trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. D. Selkoe
2. M. Mańczak
3. P. Reddy
4. F. LaFerla
5. Xinglong Wang
6. T. Wyss‐Coray
7. D. Walsh
8. R. Kayed

Who trusts M. Calkins overall

1. Xieyang Xu
2. E. Tönnies
3. M. H. Yan

Papers from M. Calkins's perspective

Ordered by how strongly the researchers M. Calkins trusts endorse each paper. Same corpus, a different order for every researcher.

1. Common Structure of Soluble Amyloid Oligomers Implies Common Mechanism of Pathogenesis (Science)
2. Mitochondrial Abnormalities in Alzheimer's Disease (Journal of Neuroscience)
3. Alzheimer's disease: genes, proteins, and therapy. (Physiological Reviews)
4. Alzheimer's Disease Is a Synaptic Failure (Science)
5. Nitric oxide‐induced mitochondrial fission is regulated by dynamin‐related GTPases in neurons (EMBO Journal)
6. Impaired Balance of Mitochondria Fission and Fusion in Alzheimer Disease (Journal of Neuroscience)
7. Cyclophilin D deficiency attenuates mitochondrial and neuronal perturbation and ameliorates learning and memory in Alzheimer's disease (Nature Medicine)
8. Mitochondria are a direct site of A beta accumulation in Alzheimer's disease neurons: implications for free radical generation and oxidative damage in disease progression. (Human Molecular Genetics)
9. Impaired mitochondrial dynamics and abnormal interaction of amyloid beta with mitochondrial protein Drp1 in neurons from patients with Alzheimer's disease: implications for neuronal damage. (Human Molecular Genetics)
10. Alzheimer's disease-affected brain: Presence of oligomeric Aβ ligands (ADDLs) suggests a molecular basis for reversible memory loss (Proceedings of the National Academy of Sciences of the United States of America)

Their papers

• Impaired mitochondrial dynamics and abnormal interaction of amyloid beta with mitochondrial protein Drp1 in neurons from patients with Alzheimer's disease: implications for neuronal damage. (2011) — 783 citations
• Impaired mitochondrial biogenesis, defective axonal transport of mitochondria, abnormal mitochondrial dynamics and synaptic degeneration in a mouse model of Alzheimer's disease. (2011) — 598 citations
• The Nrf2-ARE Pathway: An Indicator and Modulator of Oxidative Stress in Neurodegeneration (2008) — 607 citations
• Identification of the NF-E2-related Factor-2-dependent Genes Conferring Protection against Oxidative Stress in Primary Cortical Astrocytes Using Oligonucleotide Microarray Analysis* (2003) — 808 citations