D. Guertin

Molecular Biology · 2004–2012

A researcher in the Science corpus: 7 papers; trusts 12 researchers and is trusted by 34 researchers here, as inferred from the citation graph.

Subfields

Whom D. Guertin trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. Dos D. Sarbassov
  2. Siraj M. Ali
  3. E. Jacinto
  4. K. Inoki
  5. David M. Sabatini
  6. R. Loewith
  7. S. Wullschleger
  8. H. Erdjument‐Bromage
  9. M. Mueckler
  10. C. Thoreen

Who trusts D. Guertin overall

  1. T. Franke
  2. David M. Sabatini
  3. M. Laplante
  4. V. Samuel
  5. Y. Sancak
  6. A. Budanov
  7. B. Manning
  8. Gerald I. Shulman
  9. R. A. Saxton
  10. Arkaitz Carracedo

Papers from D. Guertin's perspective

Ordered by how strongly the researchers D. Guertin trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Two TOR complexes, only one of which is rapamycin sensitive, have distinct roles in cell growth control. (Molecules and Cells)
  2. Phosphorylation and Regulation of Akt/PKB by the Rictor-mTOR Complex (Science)
  3. TSC2 mediates cellular energy response to control cell growth and survival. (Cell)
  4. mTOR interacts with raptor to form a nutrient-sensitive complex that signals to the cell growth machinery. (Cell)
  5. Tumor-promoting phorbol esters and activated Ras inactivate the tuberous sclerosis tumor suppressor complex via p90 ribosomal S6 kinase. (Proceedings of the National Academy of Sciences of the United States of America)
  6. Phosphorylation and functional inactivation of TSC2 by Erk implications for tuberous sclerosis and cancer pathogenesis. (Cell)
  7. Enhanced sensitivity of PTEN-deficient tumors to inhibition of FRAP/mTOR (Proceedings of the National Academy of Sciences of the United States of America)
  8. Rictor, a novel binding partner of mTOR, defines a rapamycin-insensitive and raptor-independent pathway that regulates the cytoskeleton. (Current Biology)
  9. mTOR·RICTOR Is the Ser473 Kinase for Akt/Protein Kinase B in 3T3-L1 Adipocytes* (Journal of Biological Chemistry)
  10. TOR signaling in growth and metabolism. (Cell)

Their papers