Ine Jorgensen
Molecular Biology · 2015–2017
A researcher in the Science corpus: 2 papers; trusts 2 researchers and is trusted by 3 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 73 citations into it; #10,839 of 17,773 by trust here, #10,347 by citations
Whom Ine Jorgensen trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts Ine Jorgensen overall
Papers from Ine Jorgensen's perspective
Ordered by how strongly the researchers Ine Jorgensen trusts endorse each paper. Same corpus, a different order for every researcher.
- Non-canonical inflammasome activation targets caspase-11 (Nature)
- Pro-inflammatory programmed cell death. (Trends in Microbiology)
- Reconstituted NALP1 inflammasome reveals two-step mechanism of caspase-1 activation. (Molecules and Cells)
- Caspase‐1‐dependent pore formation during pyroptosis leads to osmotic lysis of infected host macrophages (Cellular Microbiology)
- Nalp1b controls mouse macrophage susceptibility to anthrax lethal toxin (Nature Genetics)
- The inflammasome: a molecular platform triggering activation of inflammatory caspases and processing of proIL-beta. (Molecules and Cells)
- Inflammatory caspases are innate immune receptors for intracellular LPS (Nature)
- Cytoplasmic flagellin activates caspase-1 and secretion of interleukin 1β via Ipaf (Nature Immunology)
- Cytoplasmic LPS activates caspase-11: implications in TLR4-independent endotoxic shock (Science)
- The NLRC4 inflammasome receptors for bacterial flagellin and type III secretion apparatus (Nature)
Their papers
- Pyroptotic cell death defends against intracellular pathogens (2015) — 929 citations
- Programmed cell death as a defence against infection (2017) — 877 citations