S. B. Kovacs
Molecular Biology · 2017
A researcher in the Science corpus: 1 paper; trusts 4 researchers and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 18 citations into it
- Immunology — 13 citations into it
Whom S. B. Kovacs trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts S. B. Kovacs overall
Papers from S. B. Kovacs's perspective
Ordered by how strongly the researchers S. B. Kovacs trusts endorse each paper. Same corpus, a different order for every researcher.
- Inflammasome - activated gasdermin D causes pyroptosis by forming membrane pores (Nature)
- Chemotherapy drugs induce pyroptosis through caspase-3 cleavage of a gasdermin (Nature)
- Pyroptotic cell death defends against intracellular pathogens (Immunological Reviews)
- Caspase-1-induced pyroptosis is an innate immune effector mechanism against intracellular bacteria (Nature Immunology)
- Cleavage of DFNA5 by caspase-3 during apoptosis mediates progression to secondary necrotic/pyroptotic cell death (Nature Communications)
- Caspase-11 cleaves gasdermin D for non-canonical inflammasome signalling (Nature)
- GSDMD membrane pore formation constitutes the mechanism of pyroptotic cell death (EMBO Journal)
- GsdmD p30 elicited by caspase-11 during pyroptosis forms pores in membranes (Proceedings of the National Academy of Sciences of the United States of America)
- Gasdermin D is an executor of pyroptosis and required for interleukin-1β secretion (Cell Research)
- Cleavage of GSDMD by inflammatory caspases determines pyroptotic cell death (Nature)
Their papers
- Gasdermins: Effectors of pyroptosis (2017) — 1,156 citations