William J. Kaiser

Molecular Biology · Cell Death and Differentiation · 2011–2018

A researcher in the Science corpus: 4 papers; trusts 1 researcher and is trusted by 4 researchers here, as inferred from the citation graph.

Subfields

Whom William J. Kaiser trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. Michael O. Hengartner
  2. K. Hoebe
  3. Yuqiong Liang
  4. A. Strasser

Who trusts William J. Kaiser overall

  1. Junying Yuan
  2. J. Upton
  3. R. Malireddi
  4. S. Kesavardhana
  5. L. Galluzzi
  6. T. Kanneganti
  7. Vijay A. K. Rathinam
  8. D. Ofengeim
  9. Douglas R. Green
  10. Yaqiu Wang

Papers from William J. Kaiser's perspective

Ordered by how strongly the researchers William J. Kaiser trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Chemical inhibitor of nonapoptotic cell death with therapeutic potential for ischemic brain injury (Nature Chemical Biology)
  2. cIAPs Block Ripoptosome Formation, a RIP1/Caspase-8 Containing Intracellular Cell Death Complex Differentially Regulated by cFLIP Isoforms (Molecules and Cells)
  3. RIP3, an Energy Metabolism Regulator That Switches TNF-Induced Cell Death from Apoptosis to Necrosis (Science)
  4. Mixed lineage kinase domain-like protein mediates necrosis signaling downstream of RIP3 kinase. (Cell)
  5. Human inhibitor of apoptosis proteins: why XIAP is the black sheep of the family (EMBO Reports)
  6. Identification of a molecular signaling network that regulates a cellular necrotic cell death pathway. (Cell)
  7. Toll-like receptor signalling in macrophages links the autophagy pathway to phagocytosis (Nature)
  8. Distinct BH3 domains either sensitize or activate mitochondrial apoptosis, serving as prototype cancer therapeutics. (Cancer Cell)
  9. The RIP1/RIP3 Necrosome Forms a Functional Amyloid Signaling Complex Required for Programmed Necrosis (Cell)
  10. The mitochondrial phosphatase PGAM5 functions at the convergence point of multiple necrotic death pathways. (Cell)

Their papers