M. Friedrich
Genetics · 2002–2019
A researcher in the Science corpus: 2 papers; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Genetics — 30 citations into it
- Epidemiology — 8 citations into it
Whom M. Friedrich trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- Markus F. Neurath
- A. Cerutti
- G. Kaplan
- K. Maloy
- Ramnik J. Xavier
- Hailiang Huang
- W. Sandborn
- M. Saraiva
- M. Karin
- Christopher A. Hunter
Papers from M. Friedrich's perspective
Ordered by how strongly the researchers M. Friedrich trusts endorse each paper. Same corpus, a different order for every researcher.
- Novel p19 protein engages IL-12p40 to form a cytokine, IL-23, with biological activities similar as well as distinct from IL-12.
- Induction of intestinal Th17 cells by segmented filamentous bacteria (Cell)
- Blockade of interleukin 6 trans signaling suppresses T-cell resistance against apoptosis in chronic intestinal inflammation: Evidence in Crohn disease and experimental colitis in vivo (Nature Medicine)
- Interleukin-23 drives innate and T cell–mediated intestinal inflammation (Journal of Experimental Medicine)
- Interleukin-10 and the interleukin-10 receptor. (Annual Review of Immunology)
- Inflammatory bowel disease and mutations affecting the interleukin-10 receptor. (New England Journal of Medicine)
- IL-23 is essential for T cell-mediated colitis and promotes inflammation via IL-17 and IL-6.
- A microbial symbiosis factor prevents intestinal inflammatory disease (Nature)
- Treatment of murine colitis by Lactococcus lactis secreting interleukin-10. (Science)
- Regulatory T cell-derived interleukin-10 limits inflammation at environmental interfaces. (Immunity)
Their papers
- Cytokine Networks in the Pathophysiology of Inflammatory Bowel Disease. (2019) — 635 citations
- Recruitment of stem and progenitor cells from the bone marrow niche requires MMP-9 mediated release of kit-ligand. (2002) — 1,871 citations