S. Lens
Cell Biology · 2001–2010
A researcher in the Science corpus: 2 papers; trusts 1 researcher and is trusted by 2 researchers here, as inferred from the citation graph.
Subfields
- Cell Biology — 28 citations into it
- Molecular Biology — 28 citations into it; #14,861 of 17,773 by trust here, #9,600 by citations
Whom S. Lens trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts S. Lens overall
Papers from S. Lens's perspective
Ordered by how strongly the researchers S. Lens trusts endorse each paper. Same corpus, a different order for every researcher.
- Evidence that the Ipl1-Sli15 (Aurora kinase-INCENP) complex promotes chromosome bi-orientation by altering kinetochore-spindle pole connections. (Cell)
- Targeting polo-like kinase 1 for cancer therapy (Nature Reviews. Cancer)
- The small molecule Hesperadin reveals a role for Aurora B in correcting kinetochore–microtubule attachment and in maintaining the spindle assembly checkpoint (Journal of Cell Biology)
- Regulation of HP1–chromatin binding by histone H3 methylation and phosphorylation (Nature)
- Microtubules as a target for anticancer drugs (Nature Reviews. Cancer)
- BI 2536, a potent and selective inhibitor of polo-like kinase 1, inhibits tumor growth in vivo. (Current Biology)
- The conserved KMN network constitutes the core microtubule-binding site of the kinetochore. (Cell)
- A genome-wide RNAi screen identifies multiple synthetic lethal interactions with the Ras oncogene. (Cell)
- The caspase-8 inhibitor FLIP promotes activation of NF-κB and Erk signaling pathways
- Wapl controls the dynamic association of cohesin with chromatin. (Cell)
Their papers
- NF-κB Signals Induce the Expression of c-FLIP (2001) — 795 citations
- Shared and separate functions of polo-like kinases and aurora kinases in cancer (2010) — 625 citations