K. Viola

Physiology · 2003–2018

A researcher in the Science corpus: 6 papers; trusts 3 researchers and is trusted by 1 researcher here, as inferred from the citation graph.

Subfields

• Physiology — 97 citations into it; #2,114 of 2,276 by trust here, #530 by citations
• Cellular and Molecular Neuroscience — 56 citations into it

Whom K. Viola trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. D. Selkoe

Who trusts K. Viola overall

1. F. D. De Felice

Papers from K. Viola's perspective

Ordered by how strongly the researchers K. Viola trusts endorse each paper. Same corpus, a different order for every researcher.

1. Common Structure of Soluble Amyloid Oligomers Implies Common Mechanism of Pathogenesis (Science)
2. Naturally secreted oligomers of amyloid β protein potently inhibit hippocampal long-term potentiation in vivo (Nature)
3. Altered expression of synaptic proteins occurs early during progression of Alzheimer’s disease (Neurology)
4. A specific amyloid-beta protein assembly in the brain impairs memory. (Nature)
5. Early-onset behavioral and synaptic deficits in a mouse model of Alzheimer's disease. (Proceedings of the National Academy of Sciences of the United States of America)
6. Immunization reverses memory deficits without reducing brain Aβ burden in Alzheimer's disease model (Nature Neuroscience)
7. Regulation of NMDA receptor trafficking by amyloid-β (Nature Neuroscience)
8. Pathways towards and away from Alzheimer's disease (Nature)
9. Effects of secreted oligomers of amyloid β‐protein on hippocampal synaptic plasticity: a potent role for trimers (Journal of Physiology)
10. Intraneuronal Aβ42 Accumulation in Human Brain

Their papers

• Alzheimer's disease-affected brain: Presence of oligomeric Aβ ligands (ADDLs) suggests a molecular basis for reversible memory loss (2003) — 1,118 citations
• The Amyloid-β Oligomer Hypothesis: Beginning of the Third Decade (2018) — 659 citations
• Protection of synapses against Alzheimer's-linked toxins: Insulin signaling prevents the pathogenic binding of Aβ oligomers (2009) — 616 citations
• Aβ Oligomers Induce Neuronal Oxidative Stress through an N-Methyl-D-aspartate Receptor-dependent Mechanism That Is Blocked by the Alzheimer Drug Memantine* (2007) — 904 citations
• Aβ Oligomer-Induced Aberrations in Synapse Composition, Shape, and Density Provide a Molecular Basis for Loss of Connectivity in Alzheimer's Disease (2007) — 1,061 citations
• Synaptic Targeting by Alzheimer's-Related Amyloid β Oligomers (2004) — 861 citations