V. Samuel

Epidemiology · 2004–2017

A researcher in the Science corpus: 9 papers; trusts 34 researchers and is trusted by 5 researchers here, as inferred from the citation graph.

Subfields

Whom V. Samuel trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. K. Petersen
  2. Judea Pearl
  3. S. Dufour
  4. S. Weisberg
  5. D. Guertin
  6. Gerald I. Shulman
  7. D. Befroy
  8. Ann-Hwee Lee
  9. G. Targher
  10. William L. Holland

Who trusts V. Samuel overall

  1. Gerald I. Shulman
  2. Haiming Cao
  3. Max C. Petersen
  4. M. Kotas
  5. Habib Yaribeygi
  6. R. Medzhitov
  7. R. Loomba
  8. K. Petersen
  9. Christopher D. Byrne
  10. Andreas L. Birkenfeld

Papers from V. Samuel's perspective

Ordered by how strongly the researchers V. Samuel trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Mechanism of Hepatic Insulin Resistance in Non-alcoholic Fatty Liver Disease* (Journal of Biological Chemistry)
  2. Obesity is associated with macrophage accumulation in adipose tissue. (Journal of Clinical Investigation)
  3. Obesity-associated improvements in metabolic profile through expansion of adipose tissue. (Journal of Clinical Investigation)
  4. The fat-derived hormone adiponectin reverses insulin resistance associated with both lipoatrophy and obesity (Nature Medicine)
  5. The effects of rosiglitazone on insulin sensitivity, lipolysis, and hepatic and skeletal muscle triglyceride content in patients with type 2 diabetes. (Diabetes)
  6. Chronic inflammation in fat plays a crucial role in the development of obesity-related insulin resistance. (Journal of Clinical Investigation)
  7. Molecular mediators of hepatic steatosis and liver injury. (Journal of Clinical Investigation)
  8. Endocrine regulation of the fasting response by PPARalpha-mediated induction of fibroblast growth factor 21. (Cell Metabolism)
  9. TLR4 links innate immunity and fatty acid-induced insulin resistance. (Journal of Clinical Investigation)
  10. Endoplasmic reticulum stress activates cleavage of CREBH to induce a systemic inflammatory response. (Cell)

Their papers