H. Widlund
Physiology · 2002–2013
A researcher in the Science corpus: 5 papers; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Physiology — 23 citations into it
Whom H. Widlund trusts
Their strongest outbound trust, strongest first, inferred from their citations.
- D. Kelly (in Physiology)
Papers from H. Widlund's perspective
Ordered by how strongly the researchers H. Widlund trusts endorse each paper. Same corpus, a different order for every researcher.
- Peroxisome proliferator-activated receptor gamma coactivator-1 promotes cardiac mitochondrial biogenesis. (Journal of Clinical Investigation)
- Understanding the Warburg Effect: The Metabolic Requirements of Cell Proliferation (Science)
- Transcriptional regulatory circuits controlling mitochondrial biogenesis and function. (Genes & Development)
- Mitochondria and cell death: outer membrane permeabilization and beyond (Nature reviews. Molecular cell biology)
- Myc regulates a transcriptional program that stimulates mitochondrial glutaminolysis and leads to glutamine addiction (Proceedings of the National Academy of Sciences of the United States of America)
- Faculty Opinions recommendation of PGC-1alpha-responsive genes involved in oxidative phosphorylation are coordinately downregulated in human diabetes.
- PGC-1 coactivators: inducible regulators of energy metabolism in health and disease. (Journal of Clinical Investigation)
- Transcriptional co-activator PGC-1α drives the formation of slow-twitch muscle fibres (Nature)
- Distinct sets of genetic alterations in melanoma. (New England Journal of Medicine)
- Attenuation of LDH-A expression uncovers a link between glycolysis, mitochondrial physiology, and tumor maintenance. (Cancer Cell)
Their papers
- Integrative genomic analyses identify MITF as a lineage survival oncogene amplified in malignant melanoma (2005) — 1,567 citations
- PGC1α Expression Defines a Subset of Human Melanoma Tumors with Increased Mitochondrial Capacity and Resistance to Oxidative Stress (2013) — 624 citations
- BRAF mutations are sufficient to promote nevi formation and cooperate with p53 in the genesis of melanoma. (2005) — 692 citations
- Oncogenic BRAF regulates oxidative metabolism via PGC1α and MITF (2013) — 781 citations
- Bcl2 regulation by the melanocyte master regulator Mitf modulates lineage survival and melanoma cell viability. (2002) — 779 citations