Diastolic heart failure--abnormalities in active relaxation and passive stiffness of the left ventricle.

By M. Zile, C. Baicu, W. Gaasch. New England Journal of Medicine, 2004

1,533 citations recorded by the source corpus.

Subfields: Cardiology and Cardiovascular Medicine, Radiology, Nuclear Medicine and Imaging

View Paper: https://www.semanticscholar.org/paper/b6f49f56e1c85091a898a7454bcc89e738786cc4

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In Cardiology and Cardiovascular Medicine

In Radiology, Nuclear Medicine and Imaging

Citations in the corpus

Systolic and diastolic heart failure in the community. (2006, neutral): “Atrial fibrillation/flutter 173 (31) 95 (31) 78 (32) .”
How to diagnose diastolic heart failure: a consensus statement on the diagnosis of heart failure with normal left ventricular ejection fraction by the Heart Failure and Echocardiography Associations of the European Society of Cardiology. (2007, background): “Invasively acquired evidence of diastolic LV dysfunction continues to be considered as providing definite evidence of HFNEF.(1,19,93,94) Such evidence consists of a time constant of LV relaxation (t) .”
Left ventricular heart failure and pulmonary hypertension (2015, background): “It has been shown that LV stiffness is a reliable characteristic in patients with HFpEF.50,51 An alternative to the assessment of invasive haemodynamics can be comprehensive echocardiography, which—in combination with natriuretic peptides—is feasible to diagnose HFpEF as proposed by a consensus stat”
Heart failure with preserved ejection fraction: pathophysiology, diagnosis, and treatment. (2011, background): “HFpEF, chamber size and EF are normal, and the principal haemodynamic derangement is an elevation in filling pressures.(26) When pressures are high and congestion is present at rest, HFpEF is”
Myocardial matrix remodeling and the matrix metalloproteinases: influence on cardiac form and function. (2007, background): “Indeed, recent clinical evidence suggests that progressive ECM accumulation and diastolic dysfunction are important underlying pathophysiological mechanisms for heart failure in patients with POH (196, 544, 545).”
A novel paradigm for heart failure with preserved ejection fraction: comorbidities drive myocardial dysfunction and remodeling through coronary microvascular endothelial inflammation. (2013, positive): “These studies revealed speci ﬁ c alterations in myocardial structure and function that were relevant to concentric LV remodeling and diastolic LV dysfunction characteristically observed in patients with HFPEF (9,10).”