Johanna Napetschnig
Immunology · 2012–2013
A researcher in the Science corpus: 2 papers; trusts 3 researchers and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Immunology — 32 citations into it
- Molecular Biology — 32 citations into it
- Cancer Research — 20 citations into it
Whom Johanna Napetschnig trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts Johanna Napetschnig overall
Papers from Johanna Napetschnig's perspective
Ordered by how strongly the researchers Johanna Napetschnig trusts endorse each paper. Same corpus, a different order for every researcher.
- Structural Basis of Toll-Like Receptor 3 Signaling with Double-Stranded RNA (Science)
- IRAK-4: A novel member of the IRAK family with the properties of an IRAK-kinase (Proceedings of the National Academy of Sciences of the United States of America)
- Crystal structure of the TLR1-TLR2 heterodimer induced by binding of a tri-acylated lipopeptide. (Cell)
- Activation of IKK by TNFalpha requires site-specific ubiquitination of RIP1 and polyubiquitin binding by NEMO. (Molecules and Cells)
- TAB2 and TAB3 activate the NF-kappaB pathway through binding to polyubiquitin chains. (Molecules and Cells)
- Missing pieces in the NF-kappaB puzzle. (Cell)
- The tumour suppressor CYLD negatively regulates NF-κB signalling by deubiquitination (Nature)
- Recruitment of the linear ubiquitin chain assembly complex stabilizes the TNF-R1 signaling complex and is required for TNF-mediated gene induction. (Molecules and Cells)
- Toll-like receptors. (Critical Care Medicine)
- Recognition of lipopeptide patterns by Toll-like receptor 2-Toll-like receptor 6 heterodimer. (Immunity)
Their papers
- The RIP1/RIP3 Necrosome Forms a Functional Amyloid Signaling Complex Required for Programmed Necrosis (2012) — 1,183 citations
- Molecular Basis of NF-κB Signaling (2013) — 896 citations