Toll-like receptors.

By H. S. Warren. Critical Care Medicine, 2005

4,658 citations recorded by the source corpus.

View Paper: https://www.semanticscholar.org/paper/3001979b2583e54db8341ff23db955be6208706b

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In Immunology

In Microbiology

Citations in the corpus

Role of stomata in plant innate immunity and foliar bacterial diseases. (2008, background): “PAMPs are such molecules, and they are best known for their ability to stimulate innate immunity in plants and animals (37,102).”
The delivery of therapeutic oligonucleotides (2016, background): “TLRs are most highly expressed in macrophages and dendritic cells but many cell types express at least one member of this receptor family (111).”
Toll-like receptors and innate immunity (2006, background): “CD14 concentrates LPS for binding to the TLR4/MD2 complex [8].”
“Spicing Up” of the Immune System by Curcumin (2007, background): “Toll-like receptors (TLRs) are, type I transmembrane proteins that are key regulators of innate and adaptive immune responses in mammals that can recognize distinct pathogen-associated molecular signatures (100).”
Impact of Protein Kinase PKR in Cell Biology: from Antiviral to Antiproliferative Action (2006, background): “The TLR family consists of more than 10 members, which have key roles in activating the innate immune response (343).”
Mechanisms of mesenchymal stem/stromal cell function (2016, background): “This effect was initially described ex vivo using macrophage cultures stimulated with TLR ligands such as lipopolysaccharide (LPS), zymozan, or polyinosine-polycytidylic acid (poly I:C); these simulate the effects of bacterial or viral infection [47, 48].”
Toll-like receptor function and signaling. (2006, background): “Among PRRs, a group of transmembrane proteins, Tolllike receptors (TLRs), are featured by their potent immunoadjuvant ability to activate antigen-presenting cells (APCs).(2,3) TLRs include 10 (TLRs 1-10) and 12 (TLRs 1-9 and 11-13) family members in human subjects and mice, respectively (Fig 1).”
Interferon signalling network in innate defence (2006, background): “…by the host revealed that the induction of IFNα / β genes are mediated by the activation of pattern recognition receptors (PRRs) including TLRs (Takeda et al ., 2003; Akira and Takeda, 2004; Takeda and Akira, 2005; Kawai and Akira, 2006) and retinoic acidinducible gene-I (RIG-I) and…”