E. Cario
Immunology · 2000–2007
A researcher in the Science corpus: 3 papers; trusts 1 researcher and is trusted by 4 researchers here, as inferred from the citation graph.
Subfields
- Immunology — 14 citations into it; #605 of 2,160 by trust here, #1,083 by citations
- Cancer Research — 7 citations into it
Whom E. Cario trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts E. Cario overall
- M. Abreu
- K. Honda
- Dan R. Littman
- S. Rakoff-Nahoum
- Céline Vaure
- K. Takeda
- Markus F. Neurath
- W. Strober
- Misako Matsumoto
Papers from E. Cario's perspective
Ordered by how strongly the researchers E. Cario trusts endorse each paper. Same corpus, a different order for every researcher.
- Tlr4: central component of the sole mammalian LPS sensor. (Current Opinion in Immunology)
- Bacterial Lipopolysaccharide Activates NF-κB through Toll-like Receptor 4 (TLR-4) in Cultured Human Dermal Endothelial Cells (Journal of Biological Chemistry)
- Cutting Edge: Heat Shock Protein 60 Is a Putative Endogenous Ligand of the Toll-Like Receptor-4 Complex1 (Journal of Immunology)
- Cutting Edge: Endotoxin Tolerance in Mouse Peritoneal Macrophages Correlates with Down-Regulation of Surface Toll-Like Receptor 4 Expression1 (Journal of Immunology)
- Toll-like receptors: critical proteins linking innate and acquired immunity (Nature Immunology)
- Association of NOD2 leucine-rich repeat variants with susceptibility to Crohn's disease (Nature)
- TLR4 mutations are associated with endotoxin hyporesponsiveness in humans (Nature Genetics)
- Toll-like receptor 4 imparts ligand-specific recognition of bacterial lipopolysaccharide. (Journal of Clinical Investigation)
- The repertoire for pattern recognition of pathogens by the innate immune system is defined by cooperation between toll-like receptors. (Proceedings of the National Academy of Sciences of the United States of America)
- A frameshift mutation in NOD2 associated with susceptibility to Crohn's disease (Nature)
Their papers
- Toll-like receptor 2 controls mucosal inflammation by regulating epithelial barrier function. (2007) — 696 citations
- Lipopolysaccharide Activates Distinct Signaling Pathways in Intestinal Epithelial Cell Lines Expressing Toll-Like Receptors1 (2000) — 808 citations
- Differential Alteration in Intestinal Epithelial Cell Expression of Toll-Like Receptor 3 (TLR3) and TLR4 in Inflammatory Bowel Disease (2000) — 1,354 citations