J. Christiansen
Molecular Biology · with Rajasekaran · 2006
A researcher in the Science corpus: 1 paper; trusts 3 researchers and is trusted by 4 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 8 citations into it; #7,272 of 17,773 by trust here, #12,227 by citations
- Oncology — 6 citations into it; #470 of 1,698 by trust here, #1,267 by citations
- Cell Biology — 5 citations into it; #576 of 2,073 by trust here, #1,333 by citations
Whom J. Christiansen trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts J. Christiansen overall
- Peter Friedl
- D. Sarrió
- Stephanie Alexander
- G. Moreno-Bueno
- A. Cano
- Angeliki Voulgari
- S. Rodríguez-Pinilla
- P. Paterlini-Bréchot
- H. Peinado
Papers from J. Christiansen's perspective
Ordered by how strongly the researchers J. Christiansen trusts endorse each paper. Same corpus, a different order for every researcher.
- Intravital imaging of cell movement in tumours (Nature Reviews. Cancer)
- Regulation of the Polarity Protein Par6 by TGFß Receptors Controls Epithelial Cell Plasticity (Science)
- Regulation of tight junctions during the epithelium-mesenchyme transition: direct repression of the gene expression of claudins/occludin by Snail (Journal of Cell Science)
- The two-handed E box binding zinc finger protein SIP1 downregulates E-cadherin and induces invasion. (Molecules and Cells)
- Twist, a master regulator of morphogenesis, plays an essential role in tumor metastasis. (Cell)
- Tumour-cell invasion and migration: diversity and escape mechanisms (Nature Reviews. Cancer)
- Complex networks orchestrate epithelial–mesenchymal transitions (Nature reviews. Molecular cell biology)
- Molecular requirements for epithelial-mesenchymal transition during tumor progression. (Current Opinion in Cell Biology)
- Transforming growth factor beta-1 induces snail transcription factor in epithelial cell lines: mechanisms for epithelial mesenchymal transitions. (Journal of Biological Chemistry)
- Smad-dependent and Smad-independent pathways in TGF-β family signalling (Nature)